DVT CONCEPT MAP LEDESMA.pdf

concept map on deep vein thrombosis
Virchows triad

1. LEGEND
RISK FACTORS
OF OUR PT
S/SX IN OUR
PT
INTERVENTION
Clot formation at
lower extremities
Destruction of vein
valve by clot
MP released from cancer cells
can serve as direct and indirect
contributors to the prothrombotic
mechanism
HYPERCOAGULABLE
STATE

Cancer-associated
fibroblasts express
podoplanin activation and
aggregation of platelets
VESSEL
INJURY
VENOUS
STASIS
VIRCHOW’S
TRIAD
MALIGNANCY
PREVIOUS HX OF DVT
SEDENTARY LIFESTYLE
Venous Insufficiency
Clot prevents blood
from returning to the
heart
Blood accumulating in
the leg
UNILATERAL LEG SWELLING
VENOUS INFLAMMATION
ERYTHEMA, WARMTH ,
TENDERNESS

Clot embolizes to
lungs
Pulmonary Embolism
Decrease muscle motion leads
to decreased venous blood
flow
Exposed tissue
factor in
damaged cells ,
subendothelium
leads to vwf
binding
Low blood flow leads to
increased Concentration
of blood clotting factors in
areas with vessel injury
Increased ability of
blood to coagulate
upon stimulation
UNINTENTIONAL
WEIGHT LOSS
CDS ELASTIC BANDAGE