LARYNX.pptx

Areebarajput7 45 views 23 slides Nov 02, 2022
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About This Presentation

(LAYR-inx) The area of the throat containing the vocal cords and used for breathing, swallowing, and talking. Also called voice box.


Slide Content

LARYNX RIDA HAJRA

The Larynx The larynx is an organ of voice extending from root of tongue to trachea. It consists of a complex arrangement of muscles, cartilages, membranes and ligaments. It extends from C3 to C6 in the midline (adult).

Relations Anterior – superficial structure, is covered by the fascia (deep and superficial), platysma and skin Posterior – pharynx, prevertebral muscles and cervical vertebrae Superior – pharynx Inferior – becomes continuous with the trachea

Structure 1. Hyoid bone (at C3) – not strictly part of the larynx but firmly attached above it 2. Cartilages (nine) – three unpaired and six paired: Epiglottis (elastic) – ‘leaf’-shaped; the lower, narrower end is attached to the thyroid cartilage by the thyro-epiglottic ligament, and the upper broader end is free to project superiorly Thyroid cartilage (hyaline) – like a ‘shield’. It is the largest of the laryngeal cartilages and a midline structure. Upper (at C4) and lower (at C5) borders carry cornua (horns) posteriorly – inferior cornu also has a facet for articulation with the cricoid cartilage Cricoid cartilage (hyaline) – ‘signet ring’-shaped and situated at the C6 level. It articulates on its lateral border with the thyroid cornua, and on its upper border with the arytenoid cartilages (paired)

Structure (cont.) Arytenoid cartilages (paired) – pyramidal in shape, each with a lateral muscular process (for insertion of both crico-arytenoid muscles) and an anterior vocal process (being the posterior attachment of the vocal ligament) Corniculate cartilages (paired) – two small conical-shaped cartilages, articulate with the apices of arytenoid cartilages, give attachment to aryepiglottic folds (the fibro-elastic membrane between the epiglottis and arytenoids – lower border of which is free and forms the vestibular ligament or false cord) Cuneiform cartilages (paired) – two small rod-shaped cartilages are in aryepiglottic folds and serve to strengthen them

Structure (cont.) 3. Ligaments and Membranes : Thyrohyoid membrane – between the upper border of the thyroid and the hyoid bone. Strengthened anteriorly and laterally Hyo-epiglottic ligament – connects the hyoid bone to the lower part of the epiglottis Cricothyroid ligament –interconnects cricoid, thyroid and arytenoid cartilages; upper free margin composed almost entirely of elastic tissue forms vocal ligament on each side which form interior of the vocal folds (vocal cords) ; between the thyroid above and the cricoid below, the preferred site for cricothyrotomy Cricotracheal ligament – connects the cricoid to the first ring of the trachea

Structure (cont.) Quadrangular membrane – between epiglottis and arytenoid cartilages, thickened inferior margin form vestibular ligament which form interior of vestibular folds

Structure (cont.) 4. Muscles – three extrinsic (connect larynx to its neighbours) and six intrinsic: ● Extrinsic: Sternothyroid – depresses the larynx, connects the posterior manubrium sterni to the lateral thyroid lamina Thyrohyoid – elevates the larynx, connects the lateral thyroid lamina to the inferior greater horn of the hyoid bone Inferior constrictor – constricts the pharynx, origins from the thyroid lamina, the tendinous arch over the cricothyroid and the side of the pharynx ● Intrinsic: Posterior crico-arytenoid – opens the glottis by the abducting cords Lateral crico-arytenoid – closes the glottis by the adducting cords (principle adductors)

Structure (cont.) Interarytenoid (unpaired) – closes the glottis (especially posteriorly) by connecting the arytenoids. Some fibers become the aryepiglottic muscle laterally, which constricts the laryngeal inlet somewhat Thyro-arytenoid – relaxes the cords by shortening, thus pulling the arytenoids anteriorly Vocalis – fine adjustment of vocal cord tension (fibers come from the thyro-arytenoid) Cricothyroid – only true tensor and the only muscle that lies outside the cartilages. It works by tilting the cricoid and putting stretch on the vocal cords

Vascular supply The blood supply of the larynx is derived from branches of the thyroid arteries. The cricothyroid artery arises from the superior thyroid artery itself, the first branch given off from the external carotid artery , and crosses the upper cricothyroid membrane (CTM), which extends from the cricoid cartilage to the thyroid cartilage. The superior thyroid artery is found along the lateral edge of the CTM. Arterial : Superior laryngeal (from superior thyroid artery) – accompanies the internal branch of the superior laryngeal nerve Inferior laryngeal (from inferior thyroid artery) – accompanies the recurrent laryngeal nerve Venous – into the corresponding superior and inferior thyroid veins Lymph vessels drain into deep cervical group of nodes

Nerve supply Branches of vagus (X) nerve: ● Superior laryngeal nerve – passes deep to the internal and external carotid arteries and then divides into: External branch (small) – motor to cricothyroid Internal branch (larger) – sensory above the vocal cords ● Recurrent (inferior) laryngeal nerve – It supplies: Motor to all intrinsic muscles of the larynx (except cricothyroid) Sensory supply below the level of vocal cords by inferior laryngeal nerve

Laryngeal nerve injuries Unilateral denervation of a cricothyroid muscle causes very subtle clinical findings. Bilateral palsy of the superior laryngeal nerve may result in hoarseness or easy tiring of the voice, but airway control is not jeopardized. Unilateral paralysis of a recurrent laryngeal nerve results in paralysis of the ipsilateral vocal cord, causing deterioration in voice quality. Assuming intact superior laryngeal nerves, acute bilateral recurrent laryngeal nerve palsy can result in stridor and respiratory distress because of the remaining unopposed tension of the cricothyroid muscles. Airway problems are less frequent in chronic bilateral recurrent laryngeal nerve loss because of the development of various compensatory mechanisms (e.g, atrophy of the laryngeal musculature). Bilateral injury to the vagus nerve affects both the superior and the recurrent laryngeal nerves. Thus, bilateral vagal denervation produces flaccid, mispositioned vocal cords similar to those seen aft er administration of succinylcholine. Although phonation is severely impaired in these patients, airway control is rarely a problem.

Laryngeal nerve injuries (cont.)

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