If you don’t understand the
problem you shouldn’t be trying to
fix it!
James Robb
Consultant Orthopaedic Surgeon, Royal Hospital for Sick Children, Edinburgh
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\
rez Description of the Deviation
¢ Identify.
— The joint or segment involved.
— The timing of the problem e.g. Stance or swing.
— The plane and direction involved e.g. Sagittal plane
flexion.
— The type of abnormality e.g. | Range of motion.
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€. Description of the Deviation
Drop foot gait:
— Joint/segment = Supination of the fore-foot
— Timing = seen throughout swing phase
and at initial contact but
corrects by mid-stance
— Plane = combined sagittal and coronal
= flaccid paralysis of the Peroneii
— Type
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rez Description of the Deviation
Gage [1991] has suggested 5 attributes for
normal gait:
— Stance phase stability
— Swing phase clearance
— Adequate foot pre-positioning
— Adequate step length
— Energy conservation
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“uv Description of the Deviation
Sagittal Plane:
— Reciprocal inhibition secondary to abnormal tone
— Lower motor neurone lesion e.g... Dropped foot
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xa Muscles and the child’s growth
«Mass increases as a function of the cube.
«Strength increases as a function of the
square
This explains why many marginal walkers
go off their feet as they grow.
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¡Y 4 Institute of Motion Analysis & Research
” DUNDEE The Motor System
+ The motor system comprises
the CNS and the efferent
components of the peripheral
nervous system.
+ They are primarily
responsible for the
transmission of signals
resulting in the initiation and
maintenance of muscular
contraction.
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punoee Damage to the Motor System
Neurological disorders: leads to potential
problems with:
— Loss of selective muscle control
— Dependence on primitive patterns
— Abnormal muscle tone
— Imbalance of agonists and antagonists
— Deficient equilibrium reactions EN
= Pi)
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ve 4 /
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‘pue Damage to the Motor System
ap
AO
won
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| DUNDEE The Sensory System
« The sensory system comprises
the CNS and afferent
components of the peripheral
nervous system.
« They are responsible for
information regarding the status
of the limbs in terms of:
— Motion.
— Position sense,
— Acceleration,
— Temperature,
— Pressure etc.
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punpe@ Jamage to the Sensory System
« Damage to the sensory system in isolation
will not result in abnormal gait.
« However, it may present as a complication
with other motor disorders
— e.g. spina-bifida, peripheral neuropathies
9
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DUNDEE The Control System
¢ The CNS in
conjunction with
feedback from the
ee
A
MA motor and sensory
E systems act to control
x the timing, rate and
intensity of muscle
pl activity.
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" punpeeLJamage to the Control System
+ Movement in largely determined by three
areas in the brain and abnormalities in these
will produce a particular pattern:
e Motor cortex Spasticity
+ Cerebellum Ataxia
+ Basal ganglia Dyskinesia
a
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pue The Cardio-vascular System
e All systems need
nourishment in order
to function normally.
« This is dependent on
normal cardiac and
pulmonary function.
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oo,
¡Y _
LR Gait Deviations and
Possible Causes
« Is it a stance or swing phase problem
« Is it primary or secondary
DIN
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DUNDEE Stance or Swing?
« The first consideration is to determine
which aspects of the gait cycle are
problematic.
« Problems arise in either or both the stance
and swing phases of gait.
« The demands on the body systems are quite
different for each.
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" DUNDEE Primary or Secondary
« Primary problems are the source of the
problem e.g knee arthrodesis
+ Compensations or coping responses result
from the primary problem and can
complicate the picture e.g... abnormality at
one level might affect the next level up or
down.
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om Compensations
Primary Secondary
1. Knee arthrodesis 1. Hip circumduction
2. Spasticity of Rectus Femoris 2. Vaulting on other leg
3. Hip flexion contracture 3. Equinus of the ankle
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ES Gait Deviations
DUNDEE
+ Abnormal position or range of motion
¢ Contracture
— static
— dynamic
+ Muscle weakness
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E Trunk Sagittal
DUNDEE
+ Sagittal plane: increased tilt
Assists forward shift of the trunk
Walking with aides
Secondary to increased anterior pelvic
tilt
Excessive motion can compensate for abnormal
hip, knee or ankle movements or be a
consequence of poor balance.
ASA
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y
ee
AN
A
y 4 Institute of Motion Analysis & Research
DUNDEE Pelvis S agittal:
Anterior / posterior tilt
Hip flexor/extensor
tightness
Hip extensor weakness
Abdominal weakness
Lumbar kyphosis
Poor balance
Da
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DUNDEE Hip S agittal 5
Hip flexors vs. Extensors
Decreased flexion due to posterior pelvic tilt
+ Decreased extension due to anterior pelvic tilt
¢ Crouch gait
« Slow walking
CLI
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punoee Hip excessively flexed?
+ Increased lordosis
« Knee flexion
« Hip flexion can produce compensations
distally.
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punpeLeviations at the Knee Sagittal:
« Multidirectional joint
¢ Interaction with the trunk and pelvis
+ Excessive flexion or extension
« Quadriceps vs. Hamstrings
+ Co-contraction
+ Capsular contracture
« Quadriceps weakness
« Gastrocnemius E
+ Rectus active in swing IT
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ES Knee Joint
DUNDEE
« Effectively a hinge joint
+ Sagittal plane only
+ Anomalies of flexion/extension
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ES Knee: Stance
DUNDEE
« Excessive flexion
— Muscle weakness
— Lever arm problems at the foot
« Mal-rotation
— Femoral anteversion
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Knee extension in stance?
DUNDEE
« When
« Hyper-extension
¢ Persistent flexion
Normally extended just before initial
contact and then extends fully in stance
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Knee progression angle
DUNDEE
« Observe direction of progression in stance.
« Compare with foot progression angle in
swing.
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4 \
‘pus Knee flexion in swing?
« Reduced
e Excessive
Normal range of motion 60°
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“ punpeeK nee angle at terminal swing?
«Flexed
«Extended
Normally fully extended prior to initial
contact and then flexes 15° during loading.
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< Knee: Swing
DUNDEE
+ Inadequate motion to allow foot clearance
+ Co-spasticity of Rectus Femoris and
Hamstrings
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¡Y 4
‘ous Ankle and Foot Sagittal:
+ Triceps Surae
over action or
weakness
+ Weakness of
Tibialis
anterior
+ Over
lengthened
achilles tendon
a |
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Sn Tro.
A
ES Initial contact?
DUNDEE
«Heel
«Foot-flat
«Fore-foot
Normally initial contact is made with the
heel.
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punoee Foot plantegrade in stance
eEquinus
«Calcaneus
«Valgus
°Varus
«Neutral
Normally plantegrade until immediately before
opposite foot contact.
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Foot progression angle.
DUNDEE
+ Stance
¢ Swing
Normally 5° external
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RA attitude at the end of swing.
si
Ss
eLevel
eVarus
«Valgus
Normally level, neutral or in slight
dorsiflexion.
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“pu Ankle and foot: Equinovarus
Seen usually in hemiplegia.
Heel lies in equinus and varus
Mid-foot is supinated
Fore-foot adducted and pronated
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KA Ankle and foot: Equinovarus
¢ Medial column is short
« Deformity may be mobile or fixed
— Mobile: calcaneal osteotomy or sub-talar fusion
— Fixed: triple fusion
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R A Ankle and foot: Equinovalgus
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+ Heel lies in equinus and valgus
« Mid-foot is pronated and appears ‘broken
« Fore-foot is abducted and supinated.
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xa Ankle and foot: Equinovalgus
« Lateral column is short
Deformity may be mobile or fixed
e
— Mobile: lengthening calcaneal osteotomy or
sub-talar fusion
— Fixed: triple fusion
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Ya Ankteand-foot-
~ Gait Deviation
+ Equinovarus
— instability in stance due to excessive loading of
the lateral side of the foot and a resultant
narrow base of support
« Equinovalgus
— externally rotated foot progression angle, lever
arm deficiency for the plantarflexors and a
narrow base of support
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“punvee Deviations of the Trunk Coronal:
Increased motion
Abductor insufficiency
e Assists transfer on to stance limb
Trendelenburg and Duchenne patterns
Walking with aides
« Scoliosis may produce a fixed lateral lean
x,
CLI
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E Trendelenburg
DUNDEE
+ Contralateral pelvic drop
« Hip at risk
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ES Duchenne
DUNDEE
¢ Ipsilateral trunk lean
« Protects hip
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Ss
EES
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Deviations of the Pelvis Coronal:
DUNDEE
e Adductor vs. Abductors
+ Scoliosis
« Leg length discrepancy
x,
FI
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‘»æDeviations at the Hip Coronal:
« Abductors vs. adductors
+ Scoliosis
« Leg length discrepancy
NG
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FS
=
3
4
El
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= Institute of Motion Analysis & Research
punoee Hip adduction in Swing?
«Normal
«Excessive
*Trunk rotation
Adduction may present as pelvic obliquity
or apparent shortening.
Excessive femoral anteversion can appear
like adduction.