Pivd ppt

prolapsed of inter vetebral disc
detailed view about pivd
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1. P I V D

2. Anatomy of IV disc

3. THE IV DISC CONSISTS OF THREE DISTINCT COMPONENTS : THE CARTILAGE END PLATES NUCLEUS PULPOSUS ANNULUS FIBROSUS. THE CARTILAGE RING ARE THIN LAYERS OF HYALINE CARTILAGE BETWEEN TWO ADJACENT VETEBRAL BODIES AND DISC PROPER.

4. (NP)

5. (AF)

6. Common site : C5/C6 L4/L5 L5/S1

7. DEFINATION:

8. FACTORS: CONGENTIAL DEVELOPMENTAL POOR NUTRITION POOR HEALTH HABITS ACCUMATED TRAUMA : SPORTS,AUTOMOBILE INJURY LEAD TO WEAKING OF I.V.DISC BIOMECHANICAL FACTORS POSTURE REPITITIVE MICROTRAUMA , FLEXION (COMPRESSION INJURIES) , TENSIONAL INJURIES

9. ETIOLOGY

10. PATHOLOGY IT IS SEQUENCE OF CHANGES OCURRING IN THE DISC WHICH LEAD TO PROTRUSION OR EXTRUSION OF NP THROUGH A RENT IN THE AF. THESE CHANGES CONSISTS OF THE FOLLOWING : A) NUCLEUS DEGENERATION : degenerative changes occur in the disc before displacement of the nuclear material. these changes are: Softening of the nucleus and its fragmentation Weakening and disintegration of the posterior part of the annulus

11. B) NUCLEUS DISPLACEMENT : the nucleus is under positive pressure at all the times. When annulus become weak ,either because a small area of its entire thickness has been disintegrated spontaneously or because of the injury , the nucleus tends to bilge through the defect. This is known as disc protrusion. This tendency is greatly increased if the nucleus is degenerated and fragmented. Finally the nucleus comes out of the annulus , and the under the post” longitudinal ligament though it has not lost contact between with parent disc. This is called as disc extrusion. Once the disc is extruded it cannot go back and PLL is not strong enough to prevent the nucleus protruding further. So, the extruded disc, may loose its contact with parent disc. The sequestrated disc may come to lie behind the PLL or may become free fragments in the canal.

12. C) STAGE OF FIBROSIS : this the stage of repair. the begins alongside of degeneration . The residual nucleus pulposus becomes flattened , fibrosed , and finally undergoes calcification. At the same time, new bone formation occurs at the point where the PLL has been stripped from the vertebral body and spurformation occurs .

13. MANAGEMENT ASSESSMENT TREATMENT ASSESSSMENT : POSTURE (rigid posture , loss of curve spine( i.e flattened back in lumbar spine , posterior pelvic tilt , sciatic scoliosis may present .) MOVEMENT : trunk flexion (may be painful , flexion may produce pain .) TENDERNESS : along midline or lateral to spinous processes.

14. SPECIAL TEST IN CASE OF Cx PIVD : Foraminal compression / spurling test Distraction test ULTT(upper limb tension test) Shoulder abduction test Tinel’s sign (a)

15. (b) (c) (e) (d)

16. IN CASE OF Lx PIVD: SLR Lasegue’s test Bowstring test Prone knee bending (PKB) test Sensory impairment (over dermatomes motor weakness over myotomal distraction)

17. (d) (e)

18. TREATMENT CONSERVATIVE OPERATIVE