Secondary hyperaldosteronism

for nursing students

1. SECONDARY HYPERALDOSTERONISM ALIYA rahman

2. DEFINITION Common clinical condition occurs secondary to some cause which stimulate RAAS. It is either physiological or pathological

3. etiology Physiologic causes – Salt depletion- inadequate salt intake, excessive salt through kidney or GI Pregnancy- progesterone stimulate aldosterone production pathologic causes- Hypoperfusion of kidney – nephrotic syndrome, liver cirrhosis, CHF, diuretic therapy

4. Renal ischemia- malignant hypertension,RAS High renin production by juxta glomerular apparatus

5. DIAGNOSIS Plasma sodium level- normal or near normal Plasma potassium level – low Plasma renin and aldosterone - elevated

6. MANAGEMENT TREAT UNDERLYING CAUSE Salt depletion- normal saline infusion Nephrotic syndrome, cirrohsis of liver CHF, -SPIRONOLACTONE+ LOOP DIURETIC Renal ischemia- ACE INHIBITORS RAS- TRANSLUMINAL ANGIOPLASTY Diuretic therapy- use potassium sparing diuretics.

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