National lead for GlobalSurg Collaborative National Lead with GlobalSurg ® for FALCON trial Co- Prinicipal Investigator for NRPU grant Collaborator for European Coloproctology society, UK Colorectal Fellowship, Yonsei University, South Korea Fellow of College of Physicians & Surgeons, Pakistan (Surgery) Fellow of Higher education authority of UK (FHEA) Member of Royal College of Surgeons, England (UK) Member of College of Physicians & Surgeons, Pakistan (Surgery) Member Editorial Board, BMJ case reports since 2011-2014 Reviewer for Rawalpindi Medical Journal since 2015 Dr. Ahmad Uzair Qureshi
Bile Peritonitis
Bile Peritonitis
Bile Peritonitis
Bile Peritonitis
Bile Peritonitis
Spontaneous Bact. Peritonitis Clinical features
Spontaneous Bact. Peritonitis Diagnosis
Spontaneous Bact. Peritonitis Diagnosis 250 cells / mm 3
Spontaneous Bact. Peritonitis Diagnosis 250 cells / mm 3 Micro-organism
Primary pneumococcal peritonitis Nephrotic syndrome or cirrhosis in children Routes : Females/ Males Temp/ Vomiting / Pelvic inflammatory manifestation/ Differential Leukocyte Count: 90% PMN Exudate : Sticky & odourless
Familial Mediterranean fever (periodic peritonitis) Mutations in the MEFV (Mediterranean fever) gene Abdominal pain and tenderness, mild pyrexia, polymorphonuclear leukocytosis Role of Surgery
Tuberculous Peritonitis Mycobacterium avium - ( HIV) co-infection. The abdomen is involved in 11% of patients with extrapulmonary TB I leocaecal is the most common site of involvement ) Ascitic fluid is typically a straw-coloured exudate Protein >25–30 g/L White cells >500 mm3 Lymphocytes >40%.
Tuberculous Peritonitis Acute (may be clinically indistinguishable from acute bacterial peritonitis ) and chronic forms Abdominal pain, sweats, malaise and weight loss are frequent Ascites common, may be loculated Caseating peritoneal nodules are common Intestinal obstruction may respond to anti-tuberculous treatment without surgery