Angina Pectoris detailed information.PPT

Angina Pectoris detailed information

1. .
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2. •Angina – pain
•Pectoris – chest

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3. •Angina pectoris is a clinical syndrome usually
characterized by episodes of pain or pressure in
the anterior chest -BRUNNER 2008-
•Angina is chest pain resulting from myocardial
ischemia(inadequate supply of blood to the
myocardium) -JOYCE M B;LACK 2004-
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4. • 4,45,687 death in us in 2005(1 in every 5
deaths)
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5. •Age (≥ 55 years for men, ≥ 65 for women)
•Cigarette smoking
•Diabetes mellitus (DM)
•Dyslipidemia
•Family History of premature Cardiovascular Disease (men <55 years,
female <65 years old)
•Hypertension (HTN)
•Kidney disease (microalbuminuria or GFR<60 mL/min)
•Obesity (BMI ≥ 30
 kg/m2)
•Physical inactivity

6. •Atherosclerosis
•Pulmonary embolism (a blockage in a lung artery)
•A lung infection
•Emotion/ stress
•Digestion of a large meal
• physical exertion
•Aortic stenosis (narrowing of the heart’s aortic
valve)
•Hypertrophic cardiomyopathy .
•Pericarditis (inflammation in the tissues that
surround the heart)
•A panic attack

7. Causes: Atherosclerosis, HPN, DM, Buerger’s Disease,
Polycythemia Vera, Aortic regurgitation
Reduced coronary tissue perfusion
Decreased myocardial oxygenation
Anaerobic metabolism
Increased lactic acid production (lactic acidosis)
Chest pain
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8. •Stable angina
•Unstable angina
•Prinzmetal angina or variant angina
•Post infarction angina
•Angina decubitus
•Nocturnal angina
•Silent ischemia
•Micro vascular Angina
•Intractable or refractory angina
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9. Stable angina
•stable angina is type of chest discomfort and
associated symptoms precipitated by some
activity (running, walking, emotion etc.)
normally stable angina is relieved with rest or
nitroglycerin or both.

10. Unstable angina
•It occurs at rest (or with minimal exertion),
usually lasting >10 min.
•It is severe than stable angina
•Unstable angina also can occur with or
without physical exertion, and rest or
medicine may not relieve the pain.

11. Variant (Prinzmetal's) AnginaVariant (Prinzmetal's) Angina
•A spasm in a coronary artery causes this type of angina.
•Variant angina usually occurs while you're at rest, and the
pain can be severe.
•It usually happens between midnight and early morning.
•Medicine can relieve this type of angina.

12. Cont.,
Post infarction angina
•Occurs after MI when residual ischemia may
cause episodes of angina
Angina decubitus
•Paroxysmal chest pain occurs when client sits
or stands up

13. Cont.,
Nocturnal angina
•Frequently occurs nocturnally (may be
associated with REM stage of sleep)
Silent ischemia
•Objective evidence of ischemia (such as
electrocardiographic changes with a stress
test), but patient reports no symptoms.

14. Micro vascular Angina
•Micro vascular angina can be more severe and
last longer than other types of angina.
Medicine may not relieve this type of angina.
Micro vascular Angina or Angina Syndrome X
is characterized by angina-like chest pain, but
have different causes. The cause of Micro
vascular Angina is unknown
Intractable or refractory angina
•Severe incapacitating chest pain

15. •Quality [crushing, squeezing, pressing, burning,
strangling, tight, stabbing]
•Onset [gradual or sudden,usually during activity]
•Duration [3-5 minutes, with a range of 2-15mts
•Location [substernal or retrosternal]
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16. Radiation :
[left chest and
arm,neck,jaw,teeth,back,both shoulders,
elbows and wrists]
Associated symptoms:
[nausea,vomiting,dyspnea,& diaphoresis]
Provocation:
[exertion,activity,emotional stress,
extreme temperature, heavy meals]
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17. •Physical assessment
•ECG
•Holter monitoring
•Stress test
•Cardiac imaging
•PET
•Echocardiography
•MRI
•Cardiac catheterisation&Coronary angiography
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18. •Inspection:
–Skin color
–Neck vein distention (jugular vein)
–Respiration
–Peripheral edema
•Palpation:
–Peripheral pulses

19. •Auscultation:
–Heart sounds
–Murmurs
–Pericardial friction rub

20. Cardiac Enzymes (Cardiac Markers):Cardiac Enzymes (Cardiac Markers):
1
st
: Myoglobin
a. urine = 0 – 2mg/dL (↑within 30mins – 2hrs after MI)
b. blood = <70mg/dL
2
nd
: Troponin - (Troponin T & I)
- blood = <0.6mg/dL - ↑ within 3-6hrs after MI & remains elevated for 21
days upon onset of attack
3
rd
: Creatinine kinase (CK)
CK-MB – specific to myocardial tissue (↑within 4-6hrs & decreases to
normal within 2-3days)
•male = 12-70 mg/dL
•female = 10-55 mg/dL
4
th
: LDH (specifically LDH
1- most sensitive indicator of myocardial damage) =
45-90mg/dL - ↑within 3-4 days & remains elevated for 14 days

21. Echocardiography – uses ultrasound to assess cardiac structure
& mobility
Doppler U/S – to detect blood flow of artery & vein specifically
of lower extremities (No smoking 1hr before the test)
Holter Monitoring – portable 24hr ECG monitoring which
attempts to assess activities which precipitate dysarrhythmias &
its time of the day
MRI – Magnetic fields & radiowaves are used to detect &
define abnormalities in tissues (aorta, tumors, cardiomyopathy,
pericardial disease)

22. A,B,C.D,E is promoted to reduce manifestation
and control risk factors
•Aspirin& Anti anginal
• Beta blockers and BP control
• Cigarette and cholesterol control
•Diet and diabetes control
•Exercise and education
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23. Pharmacological managementPharmacological management
1)opiate analgesic:1)opiate analgesic:
Are used to relieve or reduce acute pain
(eg.,inj.morphine)
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24. Cont.,
2)vaso dilators:
•Nitroglycerin:
It dilates the blood vessels.
•Route- sublingual, spray,oral,topical,iv
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25. 2)Beta-Blockers: (e.g, metaprolol,atenolol)2)Beta-Blockers: (e.g, metaprolol,atenolol)
it blocks the beta adrenergic sympathetic stimulation to the
heart. the result is reduction in heart rate, slowed conduction of
impulses through conduction system, decreases BP, reduces
myocardial contractility to balances the myocardial oxygen needs.
3)calcium channel blockers:(e.g,nifidipine,amlodipine)
These agents decrease the SA node automaticity and AV node
conduction resulting in slower heart rate and decrease in strength of
heart muscle contraction, these effects decrease the work load of the
heart and also relaxes the blood vessels cause decrease in BP.
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26. Cont.,
4) Lipid lowering agents – statins: 4) Lipid lowering agents – statins:
–Simvastatin
–atorvastatin
 
5) Anti platelet / anti coagulant medication:5) Anti platelet / anti coagulant medication:
prevent platelet aggregation
•Aspirin: (75 mg)
It prevents platelet activation.
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27. Cont.,
Clopidrogel:
given to the patient who are allergic to aspirin
Heparin:
It prevents the formation of new blood clots. The
dose is based on the result of APTT value
• subcutaneous injection of LMWH to treat patient
with unstable angina. LMWH increases the risk of
bleeding so the patient is monitored for s/s of
external and internal bleeding such as low
BP,increased HR.
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28. Cont.,Cont.,
•Oxygen administration:
•Directed towards MI prevention
–Lifestyle modification (individualized regular
exercise program, smoking cessation, weight
reduction)
–Stress reduction
–Diet changes
–Avoidance of cold
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29. •PTCA & CABG
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30. 
Diet instructionsDiet instructions (low salt, low fat, low cholesterollow cholesterol, high fiber);
avoid animal fats
E.g.. White meat – chicken w/o skin, fish

Stop smoking & avoid alcohol

Activity restrictions & Rest

NTGs – max of 3doses at 5-min intervals

Advise clients to always carry 3 tablets

Store meds in cool, dry place, air-tight amber bottles &
change stocks every 6months

Inform clients that headache, dizziness, flushed face are
common side effects.