DEVELOPMENT OF PALATE.ppt

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About This Presentation

DEVELOPMENT OF PALATE


Slide Content

DEVELOPMENT OF
PALATE
DR.VARDENDRA.M
DEPT. OF ORAL PATHOLOGY

CONTENTS:
INTRODUCTION
DEVELOPMENT OF PALATE
PALATAL SHELVES ELEVATION
FUSION OF PALATAL SHELVES
APPLIED ASPECTS
CONCLUSION
REFERENCES

DEVELOPMENT OF PALATE

INTRODUCTION
•Thethreeelementsthatmakeupthepalateare:(6
th
-
9
th
wk)
i)Twolateralmaxillarypalatalshelves
ii)Primarypalateofthefrontonasalprominence

PRIMARY PALATE FORMATION:
-2 medial nasal process merge at the deeper levels to
form the intermaxillary segment.
-INTERMAXILLARY SEGMENT HAS
Labial component-philtrum
Maxilla component-alveolus
Palatal component –primary palate

PRIMARY PALATE FORMATION:

PRIMARY PALATE

SECONDARY PALATE FORMATION:
-The maxillary processes give rise to two
palatal shelves.(6
th
week)

6
th
wk
12
th
wk
Fusion of
palatal
shelves
8-9
th
wk
(Elevation of
Palatal shelves)
7
th
wk
(Tongue)

HISTOLOGIC PRESENTATION

SCHEMATIC PRESENTATION

Palatal shelf elevation
Extrinsic force concept--jaw
tongue
head position
intrinsic force concept--hydration of ground substance
mesenchymal cell activity
collagen

Fusion of Palatal Shelves
Thesiteofjunctionbetweenthe3palatal
componentsismarkedbytheincisive
papillaoverlyingtheincisivecanal

SOFT PALATE:
Posterior 1/4
th
of secondary palate–soft palate.
Soft palate—bilaminar fold of mucous membrane.
It contains-palatine aponeurisis
-five pairs of muscles
levator veli palatini
tensor veli palatini
musculus uvulae
palatopharyngeus
palatoglossus
-nerves and vessels
-palatine glands

COMPLETION OF PALATE:
The secondary palate & posterior portion of
the primary palate fuses together forming the
final palate. (12
th
week).

CLINICAL CONSIDERATIONS:
-Cleft lip & cleft palates are common defects.
-Those anterior to incisive foramen –
lateral cleft lip,
cleft upper jaw &
cleft between primary & secondary palates.

-Clefts posterior to the incisive foramen
include cleft palate & cleft uvula.
-Third category -combination of clefts lying
anterior & posterior to the incisive foramen

INCOMPLETE CLEFT LIP

BILATERAL CLEFT LIP

CLEFT LIP &PALATE AND JAW

OBLIQUE FACIAL CLEFT:

MEDIAN CLEFT

SUBMUCOSAL CLEFT:

CLEFT UVULA

CYST FORMATION

CLASSIFICATION OF CLEFTS
VARIOUS TYPES:
1.Davis & Ritchie classification.
2.Veaus classification.
3.Kernahan &stark symbolic classification.
4.International confederation of plastic and
reconstructive surgery classification.

VEAU’S CLASSIFICATION OF CLEFT PALATE
GROUP1 :Defects of the softpalate only.
GROUP2 :Defects involving the hard palate
and soft palate
GROUP3 :Defects involving the soft palate to
the alveolus, usually involving the lip
GROUP4 :Complete bilateral clefts.

ETIOLOGY:
1.HERIDITORY FACTORS
2.ENVIRONMENTAL FACTORS:
Infections–measles
radiation—x-rays
hormones—cortisone
nutritional deficiencies—B-complex, vit-A,
folate metabolism,

Drugs : Ethyl alcohol
- diphenylhydantoin
Trimethadione
Retinoids
Aminopterin & methotrexate
Pr ednisolone

TRANSITION OF
BLOOD SUPPLY
Causes of cleft
lip/ palate
Deficient jaw
Growth –tongue
Occupies superior
position
Failure of
mesenchymal
cell consolidation
Lack of degeneration
of medial epithelial
seam
Failure of
elevation
Delayed shelf
elevation
Defective palatal
shelf growth

EXOSTOSIS-TORUS PALATINUS

TORUS PALATINUS

CLEFT PALATE-SYNDROMES

GOLDENHAR SYNDROME
-> Ocular, auricular,
vertebral abnormalities.

PIERRE ROBIN’S SYNDROME
->Cleft palate.
->Glossoptosis.
->Micrognathia.

MEDIAN CLEFT FACE SYNDROME
->Hypertelorism.
->Median cleft of premaxilla,
palate

ORO FACIAL DIGITAL SYNDROME
->Cleft tongue.
->Cleft of mandibular
alveolar process.

APERTS SYNDROME
->Prognathicmandible.
->Hypoplasticmaxilla.
->High arched palate.
->Parrot beak appearance.
->Hypertelorism.

TREACHER-COLLINS SYNDROME
->Hypoplasiaof mandible
and malarbones.
->Macrostomia.
->Malformation of external ear.
->High arched palate.
->Bird or fish appearance.

CROUZON SYNDROME
->Prognathicmandible.
->Hypoplasticmaxilla.
->High arched palate.
->Parrot beak appearance.
->Hypertelorism.

MARFANS SYNDROME
->Long extremities
->Hyperextensibilityof joints
->Spidery fingers
->Bifid uvula
->Cvscomplications

DOWNS SYNDROME
->Macroglossia.
->Flat face ,flat occipit.
->Large anterior fontanelle.
->Broad short neck
->Outward slanting of
palpebralfissures

CONCLUSION

REFERENCES
T.w.sadler , ed8,langman’s medical embryology, pg
no.s 272-278
Marybathbalogh, ed2, illustrated, dental embryology,
histology&anatomy.
James avery,ed3,oral histology and embryology,pg
no.s 45-49,51-61.
Tencate’s,ed7,oral histology, development, structure,
and function pg no.s 39-56.

Berkovitz, ed3,oral anatomy histology and
embryology,pgno.s-8-12,269-283.
Shafer’s,ed6,textbook of oral pathology, pg no.s-22-25.
Neville ,damm,allen,bouquot,ed3,oral and maxillofacial
pathology,pgno.s-1-5,21-22.
orban’s,ed12,textbook of oral histology,embryologypg
no.s-9&10.
Moore persaud,ed7,the development of human,pgno.s-
230-235
Profit,ed4,contemporary orthodontics,pgno.s-44-47.

THANK YOU..