Practical Ophthalmology Review Dr. Zainab Kadhim Fahad
Mature nuclear sclerotic cataract
D ense corticular cataract
H ypermature cataract with wrinkling of the anterior capsule
Morgagnian cataract with liquefaction of the cortex and inferior sinking of the nucleus
Diabetic snowflake cataract
P osterior subcapsular cataract spokes assuming a stellate morphology in myotonic dystrophy
S hield-like anterior subcapsular cataract in atopic dermatitis
B lunt trauma showing a flower-shaped opacity
Preseptal cellulitis. (A) Left preseptal cellulitis resulting from an infected eyelid abrasion; (B) axial CT showing opacification anterior to the orbital septum
CT of left orbital floor blow-out fracture – coronal view showing a defect in the orbital floor (arrow) and the ‘tear drop’ sign due to soft tissue prolapse into the maxillary antrum
Blow-out fracture of the left medial wall and floor. (A) Defective left abduction; (B) CT coronal view showing fractures of the medial wall (arrow) and floor (arrowhead)
(A) Small unstained corneal abrasion
L arge corneal abrasion stained with fluorescein
(A) Small hyphaema
T otal hyphaema
C orneal blood staining due to sustained high intraocular pressure associated with a total hyphaema
A) Retained superficial subtarsal foreign body
B) Linear corneal abrasion stained with fluorescein
(C) Insect retained in the inner canthus
D) Recently embedded corneal foreign body
Chemical burns: Limbal ischaemia
Chemical burns: Grade 2 – corneal haze but visible iris detail – the white area at left is the reflected slit beam rather than haze alone
Chemical burns: Grade 3 – corneal haze obscuring iris details
Chemical burns: Grade 4 – opaque cornea
Lower lid haematoma with subconjunctival haemorrhage in temporal side
subconjunctival haemorrhage with no visible posterior limit
Panda eyes
(A ) Upper lid colobomas ; (B) lower lid colobomas in Treacher Collins syndrome ; ( C) Treacher Collins syndrome
Dominantly inherited blepharophimosis . The child’s grandfather and father have undergone surgery to correct the defect
Blepharophimosis , ptosis and epicanthus inversus syndrome Blepharophimosis , ptosis and epicanthus inversus syndrome (BPES ) is a complex of eyelid malformations consisting of moderate–severe symmetrical ptosis with poor levator function, telecanthus , epicanthus inversus , manifesting with small palpebral fissures. Inheritance is usually Autosomal dominant ( AD ). Treatment initially involves correction of epicanthus and telecanthus , followed later by bilateral frontalis suspension . It is also important to treat amblyopia, which is present in about 50 %.
Epicanthus inversus
Lens complications of trauma Flower-shaped cataract
Lens complications of trauma D ense traumatic cataract with ruptured anterior capsule and prolapsed lens contents
Lens complications of trauma D islocation into the vitreous in a pseudophakic eye with pseudoexfoliation (arrows show the edge of the capsule )
Lens complications of trauma D islocation into the anterior chamber
Ruptured globe showing large corneal rupture with prolapse of intraocular structures
(B) bed of a dendritic ulcer stained with fluorescein
C) margins of a dendritic ulcer stained with rose Bengal
D) geographic ulcer
E) residual subepithelial haze
(F) recurrent ulceration after a corneal graft
Eyelid oedema and purulent discharge in neonatal conjunctivitis ( ophthalmia neonatorum )
Neonatal conjunctivitis ( ophthalmia neonatorum ) Causes: Organisms acquired during vaginal delivery: C. trachomatis, N . gonorrhoeae (now rare in wealthier countries, but previously responsible for 25% of childhood blindness) and herpes simplex virus (typically HSV-2). With all of these, conjunctivitis is not uncommonly associated with severe ocular or systemic complications. C. trachomatis is the most common cause in cases involving moderate–severe conjunctival inflammation . Staphylococci are usually responsible for mild conjunctivitis. Other bacterial causes include streptococci, H. influenza and various Gram-negative organisms. Topical preparations used as prophylaxis against infection may themselves cause conjunctival irritation (chemical conjunctivitis ) Congenital nasolacrimal obstruction. Despite poor neonatal tear production, a persistently mildly watery eye with recurrent mild bacterial conjunctivitis may be secondary to a blocked tear duct.
Preseptal cellulitis. (A) Left preseptal cellulitis resulting from an infected eyelid abrasion; (B) axial CT shows opacification anterior to the orbital septum
Preseptal cellulitis Diagnosis The condition manifests with a swollen, often firm, tender red eyelid that may be very severe; however, in contrast to orbital cellulitis, proptosis and chemosis are absent, and visual acuity , pupillary reactions and ocular motility are unimpaired . The patient is often pyrexial . Imaging with MRI or CT is not indicated unless orbital cellulitis or a lid abscess is suspected, or there is a failure to respond to therapy . Treatment is with oral antibiotics such as co- amoxiclav 250–500 mg/125 mg 2–3 times daily or 875/125 mg twice daily, depending on severity. Severe infection may require intravenous antibiotics . The patient’s tetanus status should be ascertained in cases following trauma.
pterygium showing cap, head, body and Stocker line (arrow)
inflamed pterygium
pseudopterygium secondary to a chemical burn
Causes of traumatic cataract. (A) Penetrating trauma (black arrow indicates corneal penetration site and white arrow indicates a cut in the anterior capsule)
(B) intralenticular metallic foreign body (arrow showing crescentic opening in capsule)