Tendoachilles rupture and its management

rohanvakta 25,163 views 44 slides Feb 27, 2014
Slide 1
Slide 1 of 44
Slide 1
1
Slide 2
2
Slide 3
3
Slide 4
4
Slide 5
5
Slide 6
6
Slide 7
7
Slide 8
8
Slide 9
9
Slide 10
10
Slide 11
11
Slide 12
12
Slide 13
13
Slide 14
14
Slide 15
15
Slide 16
16
Slide 17
17
Slide 18
18
Slide 19
19
Slide 20
20
Slide 21
21
Slide 22
22
Slide 23
23
Slide 24
24
Slide 25
25
Slide 26
26
Slide 27
27
Slide 28
28
Slide 29
29
Slide 30
30
Slide 31
31
Slide 32
32
Slide 33
33
Slide 34
34
Slide 35
35
Slide 36
36
Slide 37
37
Slide 38
38
Slide 39
39
Slide 40
40
Slide 41
41
Slide 42
42
Slide 43
43
Slide 44
44

About This Presentation

Achilles tendon is the strongest tendon of body. There are many causes of its rupture. It can be acute or chronic rupture. Management of chronic rupture by semitendinosus tendon is mentioned here.


Slide Content

Tendoachilles rupture: Management Dr Rohan Vakta M.S.Ortho AASH Arthroscopy Center Ahmedabad,India

Largest tendon in the body Origin from gastrocnemius and soleus muscles Insertion on calcanealtuberosity Anatomy

Remarkable response to stress Exercise induces increase in tendon diameter Inactivity causes rapid atrophy Age-related decreases in cell density& collagen Older athletes have higher injury susceptibility Physiology

Gastrocnemius-soleus-Achilles complex Acts on 3 joints Flexion of knee Plantarflexion of tibiotalar joint Supination of subtalar jt. It can transmit up to 10 times body weight through tendon when running Biomechanics

Risk factors Recreational athlete : Basketball , Volleyball , Rugby , Soccer [There may be a history of a recent increase in physical activity/training volume] Age (30‐50 years)

Obesity Diabetes Mellitus Previous tendon injury Risk factors (Cont.) Previous Steroid injections or fluoroquinolone use Inustrial Accidents

Classification of TA Rupture Acute -Athletics injuries Neglected -Degenerative Injuries Close Open

Stages of degenerative tendon injury AASH Arthroscopy Center

Repetitive microtrauma Relatively hypovascular area. Reparative process inadequate Most ruptures occur in “ Watershed area” Antecedent tendinitis/ tendinosis in 15% Pathophysiology of degenerative tendon injury

Athletic Injury Indirect : Eccentric force applied to a dorsiflexed foot ; Sudden unexpected dorsiflexion of ankle Direct : May occur as the result of direct trauma

Feels like being kicked in the leg Feeling of sudden Snap in the lower calf Acute sever pain Walk with a limp, unable to run, climb stairs, or stand on their toes Loss of plantar flexion power Acute

Degenerated Tendon Swelling , nodularity due to thickening and calcification crepitation along the tendon sheath Partial tear :- fusiform swelling

Physical Examination Normal TA Ruptured Tendon not Visible/Palpable Prone patient with feet over edge of bed Palpation of entire length of muscle-tendon unit during active and passive ROM

Thompson test: with the patient prone, squeezing the calf of the extended leg may demonstrate no passive plantar flexion of the foot if its Achilles tendon is ruptured Clinical Tests

“ Hyperdorsiflexion ” sign – With the patient prone and knees flexed to 90º, maximal passive dorsiflexion of both feet may reveal excessive dorsiflexion of the affected leg O’Brien needle test: insert a needle 10 cm proximal to the calcaneal insertion of the tendon. With passive dorsiflexion of the foot, the hub of the needle will tilt rostrally when the Achilles tendon is intact

X-Ray- Avulsion fracture at the insertion , with marked separation of fragments. Imaging

Kager’s Fat pad FHL TA

Inexpensive fast, reproducable , dynamic examination possible Best to measure thickness and gap Good screening test for complete rupture Ultrasound

ACUTE RUPTURE CHRONIC RUPTURE HEALTHY TENDON Expensive, not dynamic Better at detecting partial ruptures Staging of degenerative changes, (monitor healing) MRI MRI

Management Goals Optimize gastro- soleous strength and function Restore musculotendinous length and tension . Avoid ankle stiffness

Cast in Plantarflexion CAM Walker or cast with plantarflexion at 2 wks 2 wks Allow progressive weight-bearing in removable cast Remove cast and walk with shoe lift. Start with 2cm x 1 month, then 1cm x1 month then D/C 4 weeks Start physio for ROM exercises When WBAT and foot is plantigrade Start a strengthening program 2- 4 weeks Controversial 40% Re-Rupture rate Conservative Management

Preserve anterior paratenon bl. supply Beware of sural nerve Debride and approximate tendon ends Use 2-4 stranded locked suture technique Close paratenon separately Principles: Surgical management

Operative Treatment A: Defects of 1 cm or less Direct end to end repair without augmentation Bunnell Suture Modified Kessler Many techniques available

B: Defects 1 - 2 cm Muscle mobilization ± augmentation ( plantaris ) Can gain up to 2 cm with mobilization

No consensus on best reconstruction technique Semi-T tendon transfer Flexor hallucis longus (FHL) tendon transfer loss of great toe flexion(Not acceptable in Athletes) Others: FDL , Peroneus Brevis V-Y myotendinous lengthening ± FHL transfer C: Defects 2 - 5 cm

Case of tendoachilles rupture M/28 3 Months old injury USG : 25 mm gap , 38 mm proximal to calcaneal tuberosity

Surgical Technique Chronic rupture with fibrosed tissue

Plantaris 5 cm GAP

Semi-T Harvested

Semi-T passed through the proximal Musculo-Tendinous junction

Semi-T passed through Calcaneum

SemiT fixed to calcaneum using IF Screw

SemiT and Plantaris are sutured with distal & proximal TA using nonaborbable suture

Cast in Equinus for 3 Weeks

F’up at 6 wks & 2 Months

Defects > 5 cm SemiT Transfer ± V-Y myotendinous lengthening

Percutaneous vs. Open Less wound complications Lim et al. 33 patients 7 infections Higher re-rupture rate Wong et al. 367 repairs 12% re-rupture Bradley 12% perc vs. 0% open Greater Strength Cetti 111 patients General Consensus: Perc Less wound complications Better cosmesis General Consensus: Open Return to preinjury level Decreased calf atrophy Better motion Less re-rupture

Open Injury Extensive debridement Wound Care Plastic Coverage And Tendon Transfer

POST OP COMPLICATIONS Deep infection (1%) Fistula (3%) Skin necrosis (2%), Rerupture (2%).

Neither Patient nor the Surgeon want Second Surgery or Rerupture

Prevention of ReInjury Good conditoning and proper stretching before running Adequate warm‐up! Adequate rehabilitation Wearing appropriate and properly fittng shoes during activites also should be stressed to all athletes

Chronic Achilles tendon rupture Operative treatment when possible Acute Achilles tendon rupture Operative treatment for the young athletic higher demand patient Closed treatment for those patients with limited functional goals or medical comorbidities Functional rehabilitation when possible SUMMARY

Pateients ’ recovery depends largely on Their motivation , Focus & their desired postinjury activity

THANK YOU