VIVA HssdsdswewewasdasdasdasdaweewQE 8.pdf

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1. VIVA HQE #8

2. 20 CASES
CHEST
MANDIBLE
SKULL
NEURO (PEADS)

6. UL Fibrosis

BREASTS

e B: berylliosis

e R: radiation fibrosis

e E: extrinsic allergic alveolitis, eosinophilic pneumonia

e A:allergic bronchopulmonary aspergillosis, ankylosing spondylitis
e 5: sarcoidosis

e T: tuberculosis

e S:silicosis

13. LL Fibrosis

BAD AS

e 6: bronchiectasis

e A: aspiration pneumonia

e D: drugs; desquamative interstitial pneumonia
e A: asbestosis

e 5: scleroderma (and other collagen vascular diseases)

29. Button sequestrum

Dry Knipe de and assoc Pref rank Galo etal

A button sequestrum is snail sequesirum o devascularised bone surround! by luceney.
Although classically described in osteomyelitis and eosinophilic granuloma itis also occasionally
‘seen in fibrosarcoma and lymphoma,

Differential diagnoses
osteoid estoma

・ berulous osteomyeiis
radon necrosis

・ skoletal metastasis

・ fbrous dysplasia

・ epidermoid and dermoid cpt

・ haemangioma

Calvarial doughnut lesion

Sul a hen cr e a o y ta

Cara aug son re rouge sal fc, uso nd art
los ich may ave cena one des aná y cu inany part one al

Epidemiology

ost teen erin me and ur o may bn en

Clinical presentation

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POP

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‘amin eri ought asin rom rar doom lero one ap
syndrome’ ths ery race male aval dot ors wich mp ene
Be mary poble acre dei caren undempod tee on eee serum ains
osa LPs

Pathology

‘Aetiology
‘Tre ato of his ny remains union.

Microscopic features
he lors may show etal mesenchymal ar rs sue wi sr ary el
‘sy wth ii otura bone weal nd 00018 ee nal bare 12%,
There tabu steels peon; nd no lasmosden depot coses.
cian cats 3

Radiographic features

Plain radiograph

Tre dou sons eceur an ono lb Sand preset ie ape ui ray
ee i te grs y er graphe corpus

oups as sal weird are frauen surcundes y a ers seat
one srt doughs

・ ra e SU ager Dan eS rua ren a e sco margins ss
CO

econo eh group may oan ars of he dey of ayn ss iin te cuates of

racer Some esos may expand thou estro Done ern say
ee Ario y

38. BONY LESIONS OF MANDIBLE/
MAXILLA:

40. BONY LESIONS OF
MANDIBLE/
MAXILLA:

43. Ameloblastoma

Radiograph and CT

Multicystic ameloblastomas account for 80-90% of cases which are classically expansile "soap-
bubble" lesions, with well-demarcated borders and no matrix calcification. Resorption of adjacent
teeth and “root blunting” is often a feature. When larger it may also erode through the cortex into
adjacent soft tissues.

Unicystic ameloblastomas are well-demarcated unilocular lesions that are often pericoronal in
position. These are commonly found in the posterior mandible, particularly at the molars. They
are indistinguishable from other unilocular pericoronal lesions, such as dentigerous cysts,
ameloblastic fibromas and odontogenic keratocysts on CT.

MRI

In general, ameloblastomas demonstrate a mixed solid and cystic pattern, with a thick irregular
wall, often with solid papillary structures projecting into the lesion. These components tend to
enhance vividly which is very helpful to distinguish them from other lucent lesions of the
mandible.

46. OKC

‘They are typically seen as a solitary, radiolucent, unilocular, expansile lesion with smooth,
corticated borders 5, These cortices are often scalloped around the roots of teeth, Three-quarters
of lesions are located In the posterior mandible. When in the mandible, they typically grow along
the length of the bone with minimal buccolingual expansion. In the maxil, they expand into the
‘maxillary sinus. They average 3 cm in size,

‘Te appearance and location can vary". f associated with the crown af an uneruptedimpacted
tooth, they can mimic a dentigerous cyst. f associated withthe roots of nonsitaltooth, they can
mimic a radicular cyst. large enough, they will resorb the roots of adjacent teeth. They may
‘occasionally appear septated, making the distincion from ameloblastoma dificult

CT

Reminiscent of plan radiographic findings, but in better deal Visualised asanexpansle cystic
lesion with scalloped, wel-cortcated borders. Density of cystic contents varies with

viscosity. Cortical breach suggests possible soft tissue involvement.

MRI

‘Odontogenic keratocysts wil typically demonstrate %

+ TH: high signal due to cholesterol and keratin contents

+ Ta: heterogeneous signal

+ DWE restricts due o presence of keratin

・ T C+: peripheral enhancement but unlike ameloblastomas no enhancing nodular component

49. Septo-optic dysplasia

Clinical presentation

Clinical presentation of septo-optic dysplasias varied and most dependant on whether or not it
5 associated with schizencephaly (-50% of cases). This association is used to define two forms of

he condition"

・ not associated with schizencephaly

visual apparatus more severely affected
hypothalamic-pituitary dysfunction present in 60-80% of patients

may present as nypogiyceemia inthe neonatal period ó

‘small ptuitary gland vith hypoplastic or absent infundibulum and ectopic posterior
pituitary seen asa focus of 11 high signal intensity in the median eminence of
hypothalamus

olfactory bulbs may be absent (Kallman syndrome) ®

・ associated with schizencephaly

pti apparatus ess severe affected
conical anomalies: polymicrogyi, cortical dysplasia
maybe aetiological ferent ®

sometimes referred to as septo-optic dysplasia plus ®

in addition, a number of other associations are recognised including:

・ chombencephalosymapss ®

+ Chiari malformation ®

・ aqueductal stenosis®

CT

In addition to enlarged lateral ventricles with an absent septum pellucidum, CT may demonstrate
small optic apparatus (best seen with volumetric scanning and coronal reformats) and associated
small bony optic foramina

MRI

MB is the modality of choice for assessing septo-optic dysplasia,

+ may show a "point down” appearance of the lateral ventricular frontal horns on coronal images
+ absent septum pellucidum

・ hypoplastic pituitary stalk

・ hypoplastic optic chiasm/optic nerves and globes

61. ANSWER
1.PMF
2.Cystic fibrosis
3.OP
4.Asbestosis
5.Left LL collapse
6.Osteoporosis
circumscripta
7.Intradiploic epidermoid
8.Button sequestrum (OM)
9.Frontal sinus osteoma
10.Multiple myeloma
1.Maxillary SCC
2.Pindborg tumor
3.Odontoma
4.Ameloblastoma
5.OKC
6.SOD
7.Chiari II
8.DWV
9.Blake pouch cyst
10.JPA