19-Intestinal Obstruction 01-08,-15-08,22-08-24(3).ppt

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1. Case study

3. Intestinal Obstruction
“Never let the sun rise or set on small-bowel
obstruction”

4. Definitions
Intestinal Obstruction is defined as
partial or complete blockage of the
bowel that results in the failure of
intestinal contents to pass.

5. Intestinal obstruction is a common surgical
emergency & because of its serious nature it
demands early diagnosis & speedy relief.
1- Dynamic:- where peristalsis is working
against a mechanical obstruction ( acute or
chronic).
2- Adynamic:- this may occur in two forms:-
 a- Absent peristalsis: - paralytic ileus.
 b- Non-propulsive peristalsis: - Mesenteric
vascular occlusion.

6. Intestinal obstruction can be
classified into 2 types
Dynamic Adynamic
 Peristalsis is working against a
mechanical obstruction. It may
accrue in an acute or chronic
form. “Mechanical Obstruction”
 The obstructing lesion may be:

1.Intraluminal (Ex. impacted faeces,
foreign bodies, bezoar, gallstones)
2. Intramural (Ex. malignant or
inflammatory strictures)
3.Extramural (Ex. intraperitoneal bands
and adhesions, hernias, volvulus or
intussusception.)
Peristalsis is
absent (Ex.
Paralytic ileus) or it
may be present in
a non-propulsive
form (Ex. Pseudo-
obstruction)

7. Extramural Obstruction

8. Adynamic
Peristalsis may be absent (e.g. paralytic ileus)
It may be present in a non-propulsive form (e.g.
mesenteric vascular occlusion or
pseudo-obstruction).
 

9. Other Classifications
According to…
ONSET: Acute VS Chronic
 SITE: Small Bowel (High) VS Large
Bowel (Low)
NATURE: Simple VS Strangulated

10. Dynamic obstruction
Classifications
Classification Acc to Nature of presentation
acute
chronic
acute on chronic
subacute.

11. Classification acc to site of
obstruction
Small bowel obstructionLarge bowel obstruction

12. Classification acc to blood
supply
Strangulation Simple

13. Incidence
Site of Obstruction Cause Relative Incidences
(%)
Small intestine [85%]
Adhesions 60
Hernia 15
Tumors 15
miscellaneous 10
Large Intestine [15%]
CA colon 65
Diverticulitis 20
Volvulus 5
miscellaneous 10

15. COMMON CAUSES OF
INTESTINAL OBSTRUCTION
Adhesions-40%
Carcinoma-15%
Inflammatory-15%
Obstructed hernia-12%
Faecal impaction-8%
Pseudo-obstruction-5%
Miscellaneous-5%

18. Clinical Presentation

19. Classic quartet of intestinal
obstruction
Pain
Distension
vomiting
absolute constipation

20. HISTORY - The diagnosis of intestinal obstruction is based on its “cardinal symptoms”
of Pain, Distension, Vomiting and Absolute Constipation.
Clinical Presentation
1) DURATION - Nature of Presentation of Obstruction will be influenced by
whether the presentation is…
I.Acute Obstruction usually occurs in small bowel obstruction with sudden
onsets of severe colicky central abdominal pain, distension, with early
vomiting and constipation.
II.Chronic obstruction is usually seen in large bowel obstruction with lower
abdominal colic and absolute constipation, followed by distension.
III.In Acute on Chronic Obstruction there is a short history of distention and
vomiting against a background of pain and constipation.
IV.Subacute Obstruction implies an incomplete obstruction. Presentation
will be further influenced by whether the obstruction simple (With blood
supply is intact) or strangulated (there is interference to blood flow)

21. HISTORY - The diagnosis of intestinal obstruction is based on its “cardinal symptoms”
of Pain, Distension, Vomiting and Absolute Constipation.
Clinical Presentation
2) PAIN - The Pain of intestinal obstruction is true colic, and it is the first
symptom encountered.
Site- Centered around the umbilicus (small Bowel Colic)
Lower 1/3 of Abdomen (Large Bowel Colic)
Onsite- Sudden
Character - Colicky i.e. pain caused by spasm, intermittent.
Radiation - No Radiation. Generally Periumbilical or Suprapubic.
Associated Symptoms- None.
Timing- Small Bowel colic occurs every 2-20 minutes.
Large Bowel Colic occurs about every 30 minutes or more.
Exacerbating and Relieving Factors- Corresponds with Peristalsis
Severity- Sever.

22. HISTORY - The diagnosis of intestinal obstruction is based on its “cardinal symptoms”
of Pain, Distension, Vomiting and Absolute Constipation.
Clinical Presentation
3)VOMITING - Frequent vomiting, nature of Vomitus
depends on the level of obstruction.
I.Pyloric Obstruction vomitus is watery and acidic.
II.High Small Bowel Obstruction vomitus is
Greenish-Blue and Bile-Stained.
III.Lower Small Bowel Obstruction vomitus is foul
smelling and Brown (Faeculent Vomit)
IV.Large Bowel Obstruction vomitus is usually a late
symptom.

23. HISTORY - The diagnosis of intestinal obstruction is based on its “cardinal symptoms”
of Pain, Distension, Vomiting and Absolute Constipation.
Clinical Presentation
4) DISTENTION - The lower the site of obstruction
the more bowel there is available to distend.
 “Higher up” Bowel Obstruction is NOT
associated with distension.
 “Colon” Obstruction causes the colon to
distend around the periphery of the abdomen
and might extend into the small bowel if the
ileocaecal valve is incompetent.

24. HISTORY - The diagnosis of intestinal obstruction is based on its “cardinal symptoms”
of Pain, Distension, Vomiting and Absolute Constipation.
Clinical Presentation
5) ABSOLUTE CONSTIPATION - Develops
once the block becomes complete and the
bowel below is empty, so that neither feces
nor flatus are passed.
 Occurs Early in “lower” Large Bowel
Obstruction.
Occurs Late in “High” Small Bowel
Obstruction.

25. HISTORY - The diagnosis of intestinal obstruction is based on its “cardinal symptoms”
of Pain, Distension, Vomiting and Absolute Constipation.
Clinical Presentation
7) Late Manifestations…
Pyrexia
Respiratory Distress
Dehydration
Hypovolemic Shock
Peritonism

26. EXAMINATION
Clinical Presentation
1) INSPECTION - We Look For…
i.Surgical Scars
ii.Hernias
iii.Distention
iv.Visible Peristalsis

27. EXAMINATION
Clinical Presentation
2) PALPATION – Palpate for…

i.Masses
ii.Hernias
iii.Tenderness
Perform Rectal Exam.

28. EXAMINATION
Clinical Presentation
3) PERCUSSION – Percuss to hear any
Dullness or Resonance related to site of
obstruction.

29. EXAMINATION
Clinical Presentation
4) AUSCULTATION – Bowel Sounds are
Initially Loud and frequent→ Then as
bowel distends the sounds become more
resonant and high pitched→ Eventually
becoming Amphoric.
→

30. DEFERENTIAL DIAGNOSIS
Clinical Presentation
1) In The Small Bowel
I.Gallstone Ileus
II.TB
III.Tumor
IV.Adhesions
V.Volvulus

31. Extramural Obstruction

32. Intraluminal Obstruction

33. Intramural Obstruction

34. DEFERENTIAL DIAGNOSIS
Clinical Presentation
2) In The Large Bowel
I.Feces
II.Diverticulae
III.CA
IV.Hirshsprung’s Diseases
V.Adhesions
VI.Volvulus

36. Radiological diagnosis
Radiological diagnosis is based on a supine
abdominal film
Obstructed small bowel -straight segments
that are generally central and lie
transversely. No gas is seen in the colon.
Jejunum -valvulae conniventes

37. Ileum -featureless
Caecum-a rounded gas shadow in the right iliac
fossa.
Large bowel- haustral folds
Volvulus of the sigmoid colon -a grossly dilated
loop of colon, with or without visible haustrae
which arises from the pelvis and extends
obliquely across the spine to the upper abdomen.

38. X-RAY
Small Bowel
Obstruction is
suggested by a
“ladder” pattern, when
obstruction occurs,
both fluid and gas
collect in the intestine.
They produce a
characteristic pattern
called air-fluid levels.
The air rises above
the fluid and there is a
flat surface at the air-
fluid interface.

39. X-RAY
Distended Large
Bowel Tends to lie
peripherally and to
show the
Hustrations of the
Taenia Coli.

43. X-RAY- “Barium Follow-Through”
Patient drinks a contrast medium containing
barium sulfate. Contrast medium appears
white on x-rays, and shows the outline of the
internal lining of the bowel.
 X-ray images are taken at intervals as the
contrast moves through the intestine, (@ 0
minutes→@ 20 minutes→@ 40 minutes →
@90 minutes);
The bowel is accessed as it becomes visible.
The test is completed when the Barium is
visualized at the Caecum.

44. CT
Useful to detect…
• Lesions
• Colonic Tumors
•Hernias
•Bolus

45. CT Scan

47. Although the treatment of
specific causes of intestinal
obstruction is considered
accordingly, there are some
general principles applied.
Chronic large bowel
obstruction, slowly progressive,
and incomplete obstruction can
be investigated at some leisure.

Acute, sudden onset, complete
and obstruction with risk of
strangulation requires emergency
surgical intervention.

48. Treatment of acute intestinal
obstruction
Principles of treatment
Gastrointestinal drainage
Fluid and electrolyte replacement
Relief of obstruction, usually surgical

49. Preop
1.Gastric Aspiration via Nasogastric Tube; This
decompress the bowel and remove risk of
inhaling gastric contents during anesthesia.
2.IV Fluid replacement Give normal Saline,
Possibly Blood or Plasma if patient is shocked.

1.Antibiotic Therapy Started if Strangulation is
found or suspected.
 

50. PRINCIPLES OF SURGICAL
INTERVENTION
Management of segment at site of obstruction
The distended proximal bowel
Underlying cause of obstruction

51. Bowel is inspected and
non-viable (aka non-
functioning) bowel is
removed.
Non-Viability is determined by:
I.Loss of peristalsis
II.Loss of Sheen
III.Greenish or Black (Not
Purple; Purple may still
recover)
IV.Loss of Pulsation in
supplying vessels
Small Bowel can be
removed and anastomosis
performed with safety
because of its rich blood
supply.
Large bowel is not as
easily approachable,
where consideration must
be taken regarding the
location of the obstruction
and its relation to nearby
blood supply.
Operative

54. Pathophysiology
In obstruction, regardless of the cause of obstruction
or its acuteness of onset, the proximal bowel dilates
and develops an altered motility.
Below the obstruction, the bowel exhibits normal
peristalsis and absorption until it becomes empty,
when it contracts and becomes immobile.
Initially, proximal peristalsis is increased to
overcome the obstruction, If the obstruction is not
relieved the bowel begins to dilate causing a
reduction in peristaltic strength, ultimately resulting
in flaccidity and paralysis.

58. The distension proximal to an obstruction
is produced by two factors:
I.Gas
II.Fluid

62. StrangulationStrangulation
Strangulation is very dangerous condition and Strangulation is very dangerous condition and
demands early treatment before gangrene of the demands early treatment before gangrene of the
bowel arisesbowel arises..
6262

63. Strangulation
Strangulation is impairment of blood supply to bowel.
Signs of Strangulation
•Toxic Appearance, Rapid Pulse, Temperature drop
•Colicky pain with decreasing intermittence
•Marked Tenderness and Rigidity
•Raised WBC (mainly Neutrophils), usual with
infracted bowel.
•Shock

64. Causes of strangulation-
1.External→ Hernial Orifices Adhesions/Bands
2.Interrupted Blood Flow → Volvulus Intussusceptions
3.Increased Intraluminal Pressure → Closed-Loop
Obstruction
4.Primary → Mesenteric Infarction
Strangulation

65. Closed-loop obstruction
Strangulation
This occurs when the bowel is
obstructed at both the proximal
and distal point. There is no
early distension of the proximal
intestine.
When gangrene of the
strangulated segment is
imminent, retrograde thrombosis
of the mesenteric veins results
in distension on both sides of
the strangulated segment.
Unrelieved, this may result in
necrosis and perforation.

66. Carcinomatous stricture Carcinomatous stricture
(X) of the hepatic (X) of the hepatic
flexureflexure : :
closed-loop obstructionclosed-loop obstruction..
6666

67. Internal HerniaInternal Hernia::
The following are potential sites of internal The following are potential sites of internal
herniation (all are rare)herniation (all are rare)::
11 - -The foramen of winslowThe foramen of winslow..
22 - -A hole in the mesenteryA hole in the mesentery..
33 - -A defect in the transverse mescolonA defect in the transverse mescolon..
44 - -Defects in the broad ligamentDefects in the broad ligament..
55 - -Congenital or acquired diaphragmatic herniaCongenital or acquired diaphragmatic hernia..
66 - -Duodenal retroperitoneal fossaeDuodenal retroperitoneal fossae..
77 - -Caecal/appendiceal retroperitoneal fossaeCaecal/appendiceal retroperitoneal fossae..
88 - -Intersigmoid fossaIntersigmoid fossa..
6767

68. 6868

69. The standard treatment of an obstructed hernia is The standard treatment of an obstructed hernia is
to release the constricting agent by divisionto release the constricting agent by division . .
This should not be undertaken in cases of This should not be undertaken in cases of
herniation involvingherniation involving::
Foramen of WinslowForamen of Winslow , ,
Mesenteric defectsMesenteric defects
Paraduodenal/duodenojejunal fossaeParaduodenal/duodenojejunal fossae
as major BV run in the edge of the constriction ringas major BV run in the edge of the constriction ring . .
The distended loop in such circumstances must first The distended loop in such circumstances must first
be decompressed (minimising contamination) be decompressed (minimising contamination)
and then reducedand then reduced..
6969

70. Obstruction by Obstruction by
Adhesions and BandsAdhesions and Bands
7070

71. Adhesions
Most common cause of obstruction in the west.
Any site of peritoneal irritation results in fibrin
production, which results in adhesions between
apposed surfaces.
Only ONE adhesion may be causative of
obstruction.
There are many causes of intraperitoneal
adhesions such as Ischemic Areas, Foreign
Material, Infection, Inflammatory Conditions, and
Radiation Enteritis.

72. Adhesions
Adhesions may he classified into various types
whether they are early (fibrinous), late (fibrous) or
by the underlying etiology. From a practical
perspective, there are only two types — ‘easy’
weak ones and ‘difficult’ dense ones.
 Postoperative adhesions giving rise to intestinal
obstruction usually involve the lower small bowel.
Operations for appendicitis and gynecological
procedures are the most common; and are an
indication for early intervention.
 

73. 7373

74. 7474

75. ** **BandsBands : :
Usually only one band is culpable, this may beUsually only one band is culpable, this may be::
11 - -Congenital: - e.g. obliterated vitellointestinal Congenital: - e.g. obliterated vitellointestinal
tracttract..
22 - -String band following previous bacterial String band following previous bacterial
peritonitisperitonitis..
33 - -Portion of greater omentum usually adherent Portion of greater omentum usually adherent
to the parietesto the parietes..
7575

76. The following factors may limit adhesion formation:
I. Good surgical technique
II.Washing of the peritoneal cavity with saline to remove clots, etc.
III.Minimize contact with gauze
IV.Cover anastomosis and raw peritoneal surfaces.
V.Numerous substances have been instilled in the peritoneal
cavity to prevent adhesion formation, no single agent has been
shown to be safe and effective, and their use is not
recommended.

77. Treatment
Treatment of adhesions is initially
Conservative, but should not be prolonged
beyond 72hrs.
In such cases Laparotomy is required, only
causative adhesion should be removed;
removal of other adhesion will only cause
more adhesion formation.
If multiple adhesions must be removed the
bare area should be covered with omental
grafts.

78. Volvulus
A twisting or axial rotation of a portion of bowel
about its mesentery. When complete it forms a
closed loop of obstruction with resultant ischemia
secondary to vascular occlusion.
May be primary or secondary.
The primary form occurs secondary to congenital
malrotation of the gut, abnormal mesenteric
attachments or congenital bands.
 A secondary Volvulus, which is the more common
variety, is due to actual rotation of a piece of bowel
around an acquired adhesion or stoma.

86. 1) Volvulus Neonatorum
 Due to arrest gut rotation and narrow
mesentery of small bowel and Caecum .
 Symptoms include catastrophic onset of
repeated vomiting, rapid dehydration
and abdominal distension

87. 2) Volvulus of Small Intestine
Primary or secondary and usually in
the lower ileum
Spontaneously or secondary
Treatment consists of reduction of the
twist and directed to the underlying
cause .

88. 3) Cecal Volvulus
Primary or as a part of Volvulus Neonatorum .
A clockwise twist ·
F>M .
Acute features of obstruction .
25% has tympanic swelling in the midline or
left side of the abdomen .

89. 4) Sigmoid Volvulus
An anticlockwise twist .
 Most Common spontaneous Volvulus in
Adults.
Chronic constipation is a predisposing
factor.

90. S
ig
m
o
id
V
o
lv
u
lu
s
90

91. This is uncommon in Europe and the United States,
but more common in Eastern Europe and Africa.

it is the most common cause of large bowel
obstruction in the Black African population.
Rotation nearly always occurs in the anticlockwise
direction.
91

92. Predisposing causes of sigmoid volvulus are: Predisposing causes of sigmoid volvulus are:
1- Band of adhesions.
2- Overloaded pelvic colon.
3- Long pelvic mesocolon.
4- Narrow attachment of pelvic
mesocolon.
5- high residue diet and
constipation.
  
92

93. In Western populations, the condition is seen most
often in elderly patients with chronic constipation;
comorbidities are common and chronic psychotropic
drug use is associated with this condition.
Younger patients present earlier and the prognosis is
inversely related to the duration of symptoms.
Presentation can be classified as:
* Fulminant: sudden onset, severe pain, early
vomiting, rapidly deteriorating clinical course;
* Indolent: insidious onset, slow progressive course,
less pain, late vomiting.
93

94. Bolus Obstruction.
“Accumulation → Compaction”
I.Gallstones: Gallstone Ileus (stones enter the
intestine through a fistulous communication
between the bile duct and the GI tract)
II.Food: Bolus obstruction may occur after partial or
total gastrectomy when unchewed articles can
pass directly into small bowel
III.Bezoars: Trichobezoars (Hair Balls) and
Phytobezoar (Fruit/Vegetable Fibre).
IV.Worms: Ascaris lumbricoides may cause low small
bowel obstruction particularly in children, the
institutionalized and those near the tropics.

96. RadiographyRadiography - : - :
The characteristic radiological sign of gallstone ileus The characteristic radiological sign of gallstone ileus
isis
Rigler's triadRigler's triad::
small bowel obstruction small bowel obstruction ++ pneumobilia pneumobilia ++ atypical atypical
mineral shadow on radiograp ectopic calcified mineral shadow on radiograp ectopic calcified
gallstone, usually in the right iliac fossagallstone, usually in the right iliac fossa

Presence of two of these radiological signs has been Presence of two of these radiological signs has been
considered pathognomic of gallstone ileusconsidered pathognomic of gallstone ileus..
9696

97. 9797

98. 9898

99. 9999

100. TreatmentTreatment::
Laparatomy & the stone is milked proximally away Laparatomy & the stone is milked proximally away
from the site of impaction, the intestine is from the site of impaction, the intestine is
opened at this point and the gallstone removedopened at this point and the gallstone removed . .
If the gallstone is faceted, a careful check for other If the gallstone is faceted, a careful check for other
enteric stones should be madeenteric stones should be made . .
The region of the gall bladder should not be The region of the gall bladder should not be
exploredexplored..
100100

101. 101101

102. 22 - -FoodFood : :
Occur after partial or total gastrectomy when Occur after partial or total gastrectomy when
unchewed articles can pass directly into small unchewed articles can pass directly into small
bowelbowel..
The management is similar to that for gallstone, The management is similar to that for gallstone,
with intraluminal crushing usually successfulwith intraluminal crushing usually successful..
102102

103. 33 - -Trichobezoar and phytobezoarsTrichobezoar and phytobezoars::
TrichobezoarTrichobezoar: :
Undigested hair balls due to persistent hair Undigested hair balls due to persistent hair
chewing and sucking and may be associated with chewing and sucking and may be associated with
an underlying psychiatric abnormalityan underlying psychiatric abnormality..
103103

104. PhytobezoarsPhytobezoars::
results from a high fibre intake, inadequate results from a high fibre intake, inadequate
chewing, previous gastric surgery, chewing, previous gastric surgery,
hypochlorhydria and loss of the gastric pump hypochlorhydria and loss of the gastric pump
mechanismmechanism . .
When possible, the lesion may be kneaded into the When possible, the lesion may be kneaded into the
caecum, otherwise open removal is requiredcaecum, otherwise open removal is required..
104104

105. 55 - -WormsWorms::
Ascaris lumbricoides may cause low SBO Ascaris lumbricoides may cause low SBO
particularly in chidren, an attack frequently particularly in chidren, an attack frequently
follow initiation of anti-helminthic therapyfollow initiation of anti-helminthic therapy..

At laparatomy it may be possible to knead the At laparatomy it may be possible to knead the
tangled mass into the caecum, If not it should be tangled mass into the caecum, If not it should be
removedremoved..

105105

106. SBO due to SBO due to Ascaris lumbricoidesAscaris lumbricoides..
106106

107. Internal Hernia
Occurs where a portion of the small
intestine becomes entrapped in one of the
retroperitoneal fossae or into a congenital
mesenteric defect.
In the absence of adhesions hernia is
uncommon to cause obstruction and a
preoperative diagnosis is unusual.
 The standard treatment for a hernia is to
release the constricting agent by division.

108. Obstruction from Enteric Strictures
Small bowel strictures usually occur secondary
to Tuberculosis or Crohn’s disease.
Malignant strictures associated with lymphoma
are common, whilst carcinoma and sarcoma
are rare.
Presentation is usually Subacute or Chronic.
Standard surgical management consists of
resection and anastomosis.

109. In Crohn's disease, strictureplasty may be In Crohn's disease, strictureplasty may be
considered in the presence of short multiple considered in the presence of short multiple
stricturesstrictures..

110. Acute Intussusception
Most common in children.
 Primary or secondary to intestinal
pathology, e.g. polyp, Meckel's
diverticullum.
Ileocolic is the most common variant.
Can lead to an ischemic segment and
strangulation.

119. Adynamic Obstruction
I.Paralytic Ileus
II.Pseudo-Obstruction
III.Acute Mesenteric Ischemia

120. Paralytic ileus
Definition: A state in which there is failure of
transmission of peristaltic wave secondary to
neuromuscular failure
This will leads to signs of intestinal obstruction due to
accumulation of gas and fluid in the bowel with signs of
abdominal distension ,constipation, but NO Pain.
Varieties :
1. Post operative: - Self limiting, Lasts for 24-72 Hours
2. Infection: Peritonitis
3. Reflex ileus: as in fracture of the spine or ribs on in
retroperitoneal hemorrhage
4. Metabolic : Hypokalemia, DM
5. Drugs : Spasmolytic Drugs , Parkinson Drugs, Atropine

121. Clinical features:
-It takes clinical significance if there has
been no return of normal bowel sound
and no passage of flatus after 72 hrs of
Surgery
- Abdominal distension is marked,
Effortless Vomiting, but pain is NOT a
feature
-Radiologically: Multiple Fluid Level

122. Management :
1.General principles must be applied if the disease
takes place
2.Remove the cause
3.Relieve GI distension by decompression
4.Monitoring fluid and electrolyte balance
5.Rarely medical agents are used (AntiCholene
Esterase)
6.Laparotomy after 72 hours

123. Pseudo-Obstruction
This condition describes an obstruction,
usually of the colon, in the absence of a
mechanical cause or acute intra-
abdominal disease.
It is associated with a variety of
syndromes where there is an underlying
neuropathy and/or myopathy.

124. 1)Small intestinal pseudo-obstruction
•This condition may be primary or
secondary.
•The clinical picture consists of recurrent
subacute obstruction.
•The diagnosis is made by the exclusion
of a mechanical cause.
• Treatment consists of initial correction
of any underlying disorder.

125. 2) Colonic pseudo-obstruction.
•This may occur in an acute or a chronic
form.
•The acute form is known as Ogilvie
syndrome, presents as acute large bowel
obstruction.
• Abdominal radiographs show evidence of
colonic obstruction with marked caecal
distension being a common feature
•Perforation is a common complication.
•Treated by colonoscopic decompression

126. Thank You