Disorder of local abuse impact in public

AryanYadav924184 17 views 53 slides Oct 20, 2024
Slide 1
Slide 1 of 53
Slide 1
1
Slide 2
2
Slide 3
3
Slide 4
4
Slide 5
5
Slide 6
6
Slide 7
7
Slide 8
8
Slide 9
9
Slide 10
10
Slide 11
11
Slide 12
12
Slide 13
13
Slide 14
14
Slide 15
15
Slide 16
16
Slide 17
17
Slide 18
18
Slide 19
19
Slide 20
20
Slide 21
21
Slide 22
22
Slide 23
23
Slide 24
24
Slide 25
25
Slide 26
26
Slide 27
27
Slide 28
28
Slide 29
29
Slide 30
30
Slide 31
31
Slide 32
32
Slide 33
33
Slide 34
34
Slide 35
35
Slide 36
36
Slide 37
37
Slide 38
38
Slide 39
39
Slide 40
40
Slide 41
41
Slide 42
42
Slide 43
43
Slide 44
44
Slide 45
45
Slide 46
46
Slide 47
47
Slide 48
48
Slide 49
49
Slide 50
50
Slide 51
51
Slide 52
52
Slide 53
53

About This Presentation

This ppt is know about local abuse disorder and the think that we ignore about local abuse in day to day life Local abuse" can refer to different situations depending on the context. It might involve physical, verbal, or emotional abuse that occurs in a specific community or geographic area. Th...


Slide Content

Disorders of voice abuseDisorders of voice abuse
Dr. Abhinav Srivastava
Professor and Head
Dept. of ENT

Vocal nodulesVocal nodules
Syn: Singer’s nodules, teacher’s nodules,
screamer’s nodules, juggler’s nodules,
etc.
Defined as disorder of voice abuse
commonly seen in professional voice
users, characterized by hoarseness, vocal
fatigue and presence of pin head sized
raised lesions on both the vocal cords at
the junction of its anterior I/3 and
posterior 2/3.

EtiopathologyEtiopathology
‘Hyperkinetic voice’
Voice abuse- ‘professional voice
users’
–Increased intensity
–Altered pitch
–Long duration
Chronic cough
Reflux laryngitis (GERD)

Hyperkinetic voice
Maximum vibration at the junction of
anterior 1/3 and posterior 2/3
Trauma leading to odema and submucosal
microhaemorrhage
Epithelial hyperplasia and subepithelial
hyalinization and fibrosis
Bilateral vocal nodules in the free edge of
the cords

SymptomsSymptoms
Professional voice users
Hoarseness
Improves initially with voice rest
Voice fatigue (Phonesthenia)
Strained speech- pain in the neck/
throat

SignsSigns
Indirect laryngoscopy
Pin-head sized pearly white
projections on the free edge of the
vocal cords at the junction of
anterior 1/3 and posterior 2/3
Vocal cord movements are normal
Congestion of the cords +/-

InvestigationsInvestigations
Diagnosis usually made clinically
If ILS is difficult
–Flexible laryngoscopy
–Rigid angled laryngeal endoscopy (70°/
90°)
–Stroboscopy

Treatment- ConservativeTreatment- Conservative
Voice rest- ‘Absolute’
Treatment of local sepsis, cough,
reflux, etc.
Speech therapy
–Vocal hygiene
–Relaxing exercises
Early lesions may disappear

Surgical treatmentSurgical treatment
MICROLARYNGOSCOPY (MLS) AND
PRECISE EXCISION under GA
Advantage of microscope
–Hands free
–Illumination
–Magnification
Speech therapy and vocal hygiene should
be continued following excision- prevents
recurrence

Vocal polypVocal polyp
Defined as a disorder of voice abuse
commonly seen in professional voice
users, characterized by hoarseness
and presence of a sessile or
pedunculated mass usually arising
from one of the vocal cords

EtiologyEtiology
Hyperkinetic voice
–Professional voice users
–Sudden shouting
Allergy
Smoking/ other irritants
Reflux laryngitis
Chronic cough

PathogenesisPathogenesis
Hyperkinetic voice
Submucosal
hemorrhage
Bernoulli's
phenomenon
Hemorrhagic
polyp
Mucosal edema and
polyp formation
Organized, hyperplasia,
hyalinization- Vocal polyp

Pathological typesPathological types
Hemorrhagic polyp
Hyperplastic polyp
Hyaline polyp
Sessile
Pedunculated

SitesSites
Usually unilateral
Common site: Junction of anterior
1/3 and posterior 2/3
Can be seen in other areas like
anterior commisure
Free edge or under surface

SymptomsSymptoms
Common in females and between 30-
50 years
Professional voice users
Hoarseness
Phonesthenia
Diplophonia (double voice)
Very rarely, a large polyp may give
rise to choking spells/ stridor

SignsSigns
Indirect laryngoscopy
Sessile/ pedunculated mass
Smooth reddish/ whitish mass
Unilateral
Junction of anterior 1/3 and posterior 2/3
Polyp may move up and down during
respiration or may pop up during
phonation
Vocal cord movements- normal

InvestigationsInvestigations
Diagnosis usually made clinically
Flexible laryngoscopy
Rigid angled laryngeal endoscopy
Stroboscopy

DDDD
Laryngeal papilloma
Laryngeal carcinoma

TreatmentTreatment
Small/ sessile polyp- try
Voice rest
Speech therapy
Steroid inhalers
Large polyp
MICROLARYNGOSCOPY AND PRECISE
EXCISION under GA
Continue speech therapy and observe
vocal hygiene

Reinke’s odemaReinke’s odema
Syn:
–Bilateral diffuse polyposis
–Polypoid degeneration of vocal cords
Defined as benign condition of the vocal
cords, commonly seen in professional
voice users and is characterized by
diffuse odema of the entire length of both
vocal cords in the Reinke’s space
Reinke’s space: Subepithelial space in the
vocal cords

EtiopathologyEtiopathology
Same as chronic laryngitis

Clinical featuresClinical features
Symptoms
–Same as chronic laryngitis
Signs
–Diffuse odema of both vocal cords
–‘Spindle’ shaped swelling on the cords
–Vocal cords are mobile

TreatmentTreatment
Conservative
–Voice rest
–Speech therapy
–Treat etiological factors, if any
–Steroid inhalers
Surgical
–Microlaryngoscopy
–Vocal cord stripping
–One cord at a time to prevent adhesions
–Avoid injury to vocal ligament

Contact ulcer/ granulomaContact ulcer/ granuloma
Defined as disorder of voice abuse
characterized by hoarseness and
pain in the throat on speaking and
presence of of an ulcer on the vocal
process of one arytenoid and
granuloma on the vocal process of
other arytenoid

EtiologyEtiology
Hyperkinetic voice
Same as chronic laryngitis

PathologyPathology
Chronic trauma at the vocal process
of arytenoids—ulceration on one
side and granuloma on the other

SymptomsSymptoms
Hoarseness
Pain on speaking- throat/ neck
Phonesthenia
Sticky/ FB sensation in the throat

SignsSigns
Indirect laryngoscopy
Characteristic granuloma on the
vocal process of one arytenoid and
ulcer on the other
Granuloma fits into the ulcer on
phonation
DD: Malignancy of larynx

TreatmentTreatment
Microlaryngoscopy and excision
biopsy of the granuloma
Voice rest
Vocal hygiene
Speech therapy
Treat etiological factors, if any

ClassificationClassification
Benign
PAPILLOMA
Hemangioma
Glomus
Chondroma
Fibroma
Lymphangioma
Adenoma
Malignant
SQUAMOUS CELL
CARCINOMA
Adenocarcinoma
Carcinosarcoma
Small cell
carcinoma
Chondrosarcoma

PAPILLOMA OF THE PAPILLOMA OF THE
LARYNXLARYNX
Types
Juvenile laryngeal papillomatosis
(Children)
Solitary laryngeal papilloma (Adults)

Juvenile laryngeal papillomatosisJuvenile laryngeal papillomatosis
Syn: Recurrent respiratory
papillomatosis
Benign neoplasm of the larynx,
commonly seen in children and is
characterized by presence of
multiple warty lesions on the larynx
which may give rise to fatal
respiratory obstruction

IncidenceIncidence
Most common benign tumor of the
larynx in childhood
Approximately 1500 new cases
reported annually

EtiologyEtiology
Human papilloma virus 6 and 11- tissue specific-
targets stratified squamous epithelium of the
oropharynx, larynx, and anogenital region but not
epidermis
? Genetic predisposition
80-90% of cases present before 3 years of age
May get infected during birth if mother has genital
condylomata (Genital warts)- 50% of cases have
mothers with this!
But delivery by caesarian section does not prevent
it- ? Transplacental spread/ postnatal infection

PathologyPathology
Gross: Multiple pinkish white warty
lesions on the supraglottis and vocal
cords- may obstruct the airwary
Can occur anywhere in the respiratory
tract- but common glottis and
supraglottis
Microscopy: Finger like projections of
epithelial tumor cells with central
fibrovascular core

SymptomsSymptoms
Hoarseness
–‘Any child with hoarseness of more than
1 month duration should be diagnosed
as JLP until otherwise proved’
‘Asthma-like’ features
Stridor

SignsSigns
Hoarse voice/ harsh weak cry
ILS: Multiple pinkish warty lesions,
airway may be obscured
Inspiratory/ biphasic stridor +/-

InvestigationsInvestigations
X-ray lateral/ AP view of the neck
Chest X-ray
Flexible/ rigid laryngoscopy
Microlaryngoscopy and excision-
Biopsy
Bronchoscopy to r/o
tracheobronchial lesions

TreatmentTreatment
Various methods of treatment described
Microlaryngoscopy and excision with
microcautery- common method of treatment
Avoid trauma to adjacent areas- seeding
into raw areas can result in recurrence?
Recurrence rates very high
May need repeated such procedures to clear
the airway
Spontaneous regression may occur after
puberty

Other methods of treatment:Other methods of treatment:
Inconsistent success reportedInconsistent success reported
Cryosurgery
Ultrasonic destruction
Laser vaporization
MLS and application of podophyllum
Interferon therapy
Autogenous vaccines
Long term antibiotics have been tried
Antiviral treatment

If stridor +If stridor +
Tracheostomy if intubation is not
possible
Tracheostomy should be avoided
because recurrence in the
tracheastoma site can occur- more
difficult to treat

PrognosisPrognosis
High recurrence rates
Death due to respiratory obstruction
If survives till puberty- prognosis is
good
NOT PREMALIGNANT

SOLITARY PAPILLOMASOLITARY PAPILLOMA
Occurs in adults
Clinical presentation is similar to vocal
polyp- commonly occurs on membranous
vocal cord
Etiology: Human papilloma virus
Treatment: Microlaryngoscopy and
excision
PREMALIGNANT CONDITION
Prognosis is good if completely excised-
recurrence rates very low compared to
Juvenile papillomatosis

Thank you