GOO is the clinical and pathophysiological consequence of any symptom complex that produces a mechanical impediment to gastric emptying. HISTORY AGE :20-45 years with peak 30-35 years Abdominal pain site:epigastric and left hypochondrial pain relationship to food: food - pain -relief=du food – pain = gu
relieved by alkali,milk association with time of the day h/o radiation to the back(? Pancreas penetration) generalised pain(perforation) Anorexia,nausea Early satiety Vomiting- characteristic unpleasant -copious -projectile -non bilous,food taken several hours to days ago
Feeling of unwell Weight loss Abdominal swelling EXAMINATION Chronically ill looking Wasted,dehydrated Pale May be in shock Epigastric /left or right hypochondrial tenderness
INVESTIGATIONS To stabilise patient -complete haemogram -serum electrolytes, -arterial blood gases -urinalysis To confirm diagnosis -plain x-ray abdomen erect -gastric function tests:>400ml resting juice aspirated shows presumptive diagnosis of GOO -endoscopy and biopsy
-barium meal:findings markedly dialated stomach with a lot of residue gastritis,stasis chronic cicatrised ulcer,diverticula trifoliate deformity of duodenal cap pyloric opening narrowed or total obstruction Detection of H.pylori -Non invasive serology
carbon labelled urea breath test -invasive rapid ureasetest histology and culture
POST OP COMPLICATIONS immediate:primary haemorrhage injury to contiguous strictures aneasthetic complications early: postgastrecrtomy syn i )early dumping: 20-30 mins after ingestion ofmeal both GI and cardiovascular symptoms Mgt-pt.informed preop dietary modification,long acting somatostatin analogue,jejunal 20cm isoperistaltic loop interposition,jejunal 10cm antiperistaltic loop interposition
2) Late dumping: due to hypoglycaemia Mgt:small meals,less carbohydrates,antiperistaltic loop duodenal blow out:4-5 th post op day,life threatening, mgt;fluid and electrolyte correction,enteroentostomy