posterior cruciate ligament injury.ppt. .

AkshayBadore2 276 views 76 slides Aug 19, 2024
Slide 1
Slide 1 of 76
Slide 1
1
Slide 2
2
Slide 3
3
Slide 4
4
Slide 5
5
Slide 6
6
Slide 7
7
Slide 8
8
Slide 9
9
Slide 10
10
Slide 11
11
Slide 12
12
Slide 13
13
Slide 14
14
Slide 15
15
Slide 16
16
Slide 17
17
Slide 18
18
Slide 19
19
Slide 20
20
Slide 21
21
Slide 22
22
Slide 23
23
Slide 24
24
Slide 25
25
Slide 26
26
Slide 27
27
Slide 28
28
Slide 29
29
Slide 30
30
Slide 31
31
Slide 32
32
Slide 33
33
Slide 34
34
Slide 35
35
Slide 36
36
Slide 37
37
Slide 38
38
Slide 39
39
Slide 40
40
Slide 41
41
Slide 42
42
Slide 43
43
Slide 44
44
Slide 45
45
Slide 46
46
Slide 47
47
Slide 48
48
Slide 49
49
Slide 50
50
Slide 51
51
Slide 52
52
Slide 53
53
Slide 54
54
Slide 55
55
Slide 56
56
Slide 57
57
Slide 58
58
Slide 59
59
Slide 60
60
Slide 61
61
Slide 62
62
Slide 63
63
Slide 64
64
Slide 65
65
Slide 66
66
Slide 67
67
Slide 68
68
Slide 69
69
Slide 70
70
Slide 71
71
Slide 72
72
Slide 73
73
Slide 74
74
Slide 75
75
Slide 76
76

About This Presentation

PCL injury


Slide Content

DR. Akshay R B

Posterior cruciate ligament (PCL) tears
comprise 3% of outpatient knee injuries and
38% of acute traumatic knee hemarthroses
These injuries rarely occur in isolation, and
up to 95% of PCL tears occur in combination
with other ligament injuries

 Anatomy of PCL
 Mechanism of Injury
 Clinical Evaluation
 Investigations
 Management

Tibial footprint is located
between the posterior horns of
two menisci about 1–1.5 cm
below the posterior tibial
margin in the ‘PCL facet’

AL and PL fibers further extend
upward and medially to be
attached onto the medial
femoral condyle.
ALB is attached mostly to the
roof of the intercondylar notch
and PLB to the medial side of the
wall

More Important
in Extension of
the knee
More Important in
Flexion of the knee

The PCL is also strengthened
by the meniscofemoral
ligaments (MFL), anteriorly
(ligament of Humphrey) and
posteriorly (ligament of
Wrisberg).

Tensile loads are in the range of 2k to 3kN
38 mm in length x 13 mm in diameter
Diameter is 1.3 times larger than the ACL

Posterior Tibial Translation
Rotational and Medial/Lateral Stability
Normal Joint contact pressure

Restrains posterior tibial translation as the
knee moves from extension to flexion
throughout the arc of motion (0–120°)
especially from 30°–90° flexion
Posteromedial, posterolateral capsule and
collaterals aid in the posterior restraint
between 0° and 30° flexion

Covey et al. demonstrated that Posterior
translation of tibia increases by two fold (7.23
± 0.65 mm) at 90° flexion as compared with
20° flexion (3.41 ± 0.77 mm) at 74 N
posteriorly directed force over tibia after
selective sectioning of the PCL

The role the PCL plays in the rotational control of the knee
is still unclear, with many contradictory studies published
in the literature
It restricts internal rotation at all flexion angles, PMB
particularly was reported to be controlling rotation beyond
90º of flexion
It acts as a secondary stabilizer to rotational forces when
other ligaments are compromised and other ligaments may
provide control to rotation when the PCL is deficient

The deficiency of the PCL results in
increased joint contact pressures in the
medial and patellofemoral compartments
Untreated PCL deficiency have greater
incidence of medial and patellofemoral
compartment degeneration

Results showed significant posterior
subluxation of the tibia at 60° of flexion in the
PCL-deficient specimen, which resulted in
increased contact pressure and pressure
concentration in the medial compartment

Direct blow to proximal tibia with a flexed knee
(dashboard injury)
Hyperflexion with a plantar-flexed foot
Hyperextension injury
External rotation force on a weightbearing leg
with the knee in near full extension

History
Physical Examination

Exploring the mechanism of injury
Energy or velocity imparted to the knee
during the injury
Did the knee swell up immediately?
Could the patient bear weight? Did the knee
feel unstable?
Current symptoms including pain, stiffness,
instability

Look
Feel
Move

Posterior drawer test
Posterior sag test (godfrey test)
Quadriceps active test
Dial test
Varus/valgus stress

Performed at 90º of knee flexion, and has a
sensitivity of 90% and a specificity of 99%
Isolated PCL translate >10-12mm in neutral
and >6-8mm in internal rotation.
Combined ligamentous injuries translate
> 15mm in neutral and >10mm in internal
rotation.

Plane Radiography
Stress Radiography
Magnetic Resonance Imaging

A standard knee series, including
bilateral standing (AP),
AP flexion 45° weight bearing
Lateral and
Merchant patellar radiographs
should be evaluated for any evidence of avulsion
fractures, tibial subluxation and associated knee
injuries and chronic cartilage damage

Stress radiography has been gaining
popularity for the diagnosis of multi-
ligamentous knee injuries
It involves the application of a standardized
force to the knee to produce abnormal joint
displacement

Several techniques have been described
including hamstring contraction, gravity
assisted, the Telos device and single-leg
kneeling
The Telos device and kneeling have been
shown to be superior to other methods for
reproducibly demonstrating posterior knee
instability

A diagnostic algorithm has been validated where
side to side posterior translation difference has
been quantified
1. 0–7 mm = a partial PCL tear
2. 8–11 mm = isolated complete PCL tear
3. ≥12 mm = combined PCL and posterolateral
corner or posteromedial corner knee injury

High sensitivity (near 100%) and specificity (near
97%)
MRI is the radiologic study of choice in
diagnosing acute PCL tears, Although chronic
PCL injuries may be apparent on MRI it is not as
sensitive in diagnosing chronic tears
MRI may appear normal as soon as 3 months
following low-to moderate-grade PCL injuries

Normal PCL is homogeneously low signal on both
T2 and proton density weighted sequences,
lacking internal striations like ACL.
Normal PCL should measure 6 mm or less, when
measured from anterior to posterior in the
sagittal plane

There are two potential pitfalls if one relies
only on the sagittal plane
First, partial tears may be interpreted as
complete tears.
Second, mucoid degeneration may mimic a
PCL tear in the setting of a functionally stable
ligament

Nonoperative vs operative
Repair vs reconstruction
Autograft vs allograft
Single vs double bundle
Arthroscopic vs Open technique
Rehabilitation

Acute Isolated grade I and Grade II tears (posterior
tibal translation < 10mm)
Asymptomatic patients
Knee should be immobilized for 2-4weeks
Functional dynamic force braces have been designed
to keep the knee in anterior drawer to avoid laxity
during healing
Strengthening of quadriceps and avoiding hamstrings
use

Grade III injuries with >10 mm of posterior
tibial displacement
Symptomatic complete tears
PCL tears with other ligamentous injuries
(ACL, MCL, PLC)
Acute bony avulsion injuries of the PCL
attachment
Failure of conservative management

Long-term subjective evaluations of patients are very
comparable
At a mean of 17 years after non operative treatment,
Shelbourne et al. found a mean IKDC score of 73,
which compares to IKDC scores of 65 found by 2
nd

study of operative treatment that had much less
follow-up times of 9–10 years

Arthroscopic primary PCL repair with suture
augmentation can be performed in patients
with proximal soft tissue avulsion tears
Ligament remnants that can be re
approximated to the femoral wall and have
sufficient tissue quality to withhold sutures
can be primarily repaired rest needs
reconstruction

Autograft
Allograft
Synthetic grafts

Bone–patellar tendon–bone (B-PT-B)
Hamstring (semitendinosus and/or gracilis)
Quadriceps tendon–patellar bone (QTB)

No risk of transmission of an infectious
disease
Faster incorporation with adjacent tissues
No risk of immune-mediated tissue rejection

Achilles tendon
Double-stranded anterior and posterior tibial
tendons
Peroneus longus tendons

Eliminates donor-site morbidity
Multiple ligament injuries in which multiple
grafts will be required

Meta-analysis shows that the clinical
outcomes were similar between allograft
and autograft tendons for PCL
reconstruction

Theoretically has the advantages of availability,
consistency, and appropriate mechanical strength, no
donor site morbidity and no risk of disease transmission
Eg. Carbon fiber, dacron, bundled
polytetrafluoroethylene (GORE-TEX™), ABC carbon,
polyester
Longer term follow-up demonstrated recurrent
instability and chronic effusions hence their use is
controversial

Only AL bundle is reconstructed during
single-bundle PCL reconstruction
One femoral tunnel is made
Both auto and allografts can be used

Both ALB and PMB are reconstructed in
Double bundle PCLR
Theoretically it restore the normal
kinematics
It requires two separate femoral tunnels to
reconstruct ALB and PMB that puts femur at
risk for MFC fractures.

This systematic review found that double-
bundle reconstruction was superior to
single-bundle in biomechanical studies BUT
clinical outcomes showed no significant
differences between the two PCL
reconstruction techniques

Preferred technique is a single AL
bundle reconstruction because it
reduces surgery time and clinical
evidence demonstrates no advantage

Transtibial tunnel technique
Tibial-inlay technique

Aims to simulate the tibial and femoral ALB
origins
Incase of DB PCLR a 5-mm bone bridge is
maintained between femoral tunnels

The main concern in this technique is the so-
called ‘killer turn’, the sharp angle on the tibial
tunnel exit that may produce abrasion,
attenuation and subsequent graft failure

Proximity of neurovascular structures to the PCL
insertion is another challenge. The anterior wall
of the popliteal artery lies approximately 7–10
mm from the posterior border of the PCL at 90°
of flexion

If only ALB is reconstructed the graft is
tensioned between 70°-90° of knee flexion
For Double bundle PMB is tensioned is full
knee extension

The two strongest advantages of tibial inlay
technique are its secure bone-to-bone
fixation on the tibia, and the elimination of
the “killer turn”
Both open and arthroscopic techniques are in
practice

Phase I ( 0 – 6 weeks)
Phase II (6weeks – 6 months)
Phase III (6 Months – 12 Months)

1
st
4 weeks= brace locked in full extension, passive ROM
up to 90° flexion, NWB with crutches
After 4 weeks= brace unlocked to 100°, passive ROM
beyond 90°,weight bearing as tolerated with crutches
and brace on
Quad sets ,Straight leg raise (SLR) with brace locked,
Ankle DF and PF, avoid active contraction of hamstrings
Patella mobilization
Discontinue brace and crutches at 6 weeks

Passive stretching
Closed chain exercises as tolerated
Maximum knee flexion: 10–15° terminal
flexion deficit is not unusual
Quadriceps strength 80–90 % of the
contralateral limb

Quadriceps symmetry
Open and closed chain exercises as tolerated
Return to sport-specific activity as tolerated

They found the overall complication
rate for arthroscopic knee surgery was
4.7 %; however, 20.1 % of PCL
reconstructions had a complication

Neurovascular injury
Loss of flexion ( 10-20 degrees)
Failure to obtain objective stability
Osteonecrosis of the medial femoral condyle