Refractive State Slightly more hyperopic until age 7 Emmetropization Process in the developing eye in which the refractive power of anterior segment & axial length of the eye adjust to reach emmetropia
Vision Assessment Subjective refraction inappropriate Requires paralysis of accomodation Objective refraction be choice Presence of strabismus needs modification of prescribing
Vision Assessment
Vision Assessment Fixation and Following Behavior
Preferential Looking Test Vision Assessment
Recognition visual acuity Vision Assessment
Optometry Australia’s Paediatric Eye Care Reference Guide, September 2019 Vision Assessment
Cycloplegic Refraction Vision Assessment
Cycloplegic Refraction Vision Assessment
Refractive Error Classification 02
Congenital Myopia Developmental Mild : -0.25 - -3.00 D Moderate : -3.25 - -6.00 D Severe : > -6.00 D Focusing power too strong / AXL too long Occur as a results of variety genetic and environmental factors
Congenital Myopia Developmental Cycloplegic refraction : until 10 years, very high myopia (>10.0D) & infant with esotropia Corrected full correction include cylinder Undercorrect controversial Undercorrect in esotropia for decrease deviation rarely tolerated Overcorrection additional accommodative stress Parents education Contact lens for high myopia
Myopia Prescribing
> 5.00 D : more visually impaired Hyperopia Low hyperopia not necessary to correct Full correction cycloplegic refractive hyperopia + esotropia Reduce amount of correction in school age child + short course cycloplegia
Hyperopia Prescribing
Astigmatism Full correction Moderate or severe astigmatism might be corrected in children <3 years old to give optimal vision
Astigmatism Prescribing
Amblyopia 03
Amblyopia Prevalence 2-4% in North America Most common cause : unilateral vision impairment in adults < 60 yo Prevalence increased with : Born prematurely Developmental delay Family history
Amblyopia Unilateral or, less commonly, bilateral reduction of best- corrected visual acuity (also referred to as corrected distance visual acuity) that cannot be attributed directly to the effect of any structural abnormality of the eye or visual pathways Strabismus Refractive error Visual deprivation
Vision Assesment Acuity Developed Visual deprivation can cause amblyopia Amblyopia treament still effective Birth to 3-5 years age A few months to 7-8 years age Up to 9 years age
Pathophysiology Visual system vulnerable abnormal input Early postnatal development : critical period cortical development Visual system plasticity amblyopia revearsal Amblyopic visual deficit visual cortical changes, lost cells of primary visual cortex
Amblyopia Treatment 03
Steps 3 Promote use the amblyopic eye 1 Eliminate obstruction visual axis 2 Correct any significant refractive error
Steps Refraction correction Occlusion Cataract removal Aphakia : initiated promptly Refractive surgery Unilateral : first 6 weeks of life Bilateral : first 10 weeks of life Unilateral, follow up 2-3 months Full time occlusion follow up < 1 week per year of age in < 4 years Severe (20/125 – 20/400) part time (6 h) Moderate (> 20/100) –2 h
Binocular treatment Electronic device under dichoptic viewing condition Orthotropia Small angle strabismus Active / passive visual task (require simultaneous perception) High contrast (amblyopic eye) & low contrast (fellow eye)
Complication and Challenges in Amblyopia Therapy 03
Reverse amblyopia & new strabismus Poor adherence Unresponsiveness Recurrence 01 03 02 04 Full time occlusion Undercorrection hyperopic with cycloplegia Treatment cost adhesive / cloth patches Irritation Children older 5 years old Reexamination anomalies Terminated when lack progress over 3-6 months Reducing treatment before cessation Children 7-12 years age more sustainable Stable : 12-mo exam Complication
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Algoritma Tatalaksana Amblyopia
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