synovitis.pptx

synovitis

1. OVIUM & CRYS
OVITIS

2. SXNONIUM

+ Differentiates from the mesenchym
tissue around the articular disc

+ Clears the articular surface by the
month in utero.

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4. Bone :
, Articular (hyaline) cartilage

Synovial fluid Fibroblast

Dense

Loose fibr
connective

5. €
ve rise tot

re
gl

ip

6. SACOM IO

+ Clear viscous yellow fluid) which does not
on standing (no fibrinogen),

+ Kinge joint-O/5ml of Fluid.

+ Viscosity depends on conc of hyaluronic a

+ Lowered in ageing osteoanthnitisjand traun

# 96/0 water and 4%osalutes with alspecific le
OF 1.010) and a phio17.3+7.6.

+ Reduced in osteoarthritis and alter trauma!

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ell

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plasma,
i

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=
+

CIN IM

PONE

10. pout,
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te | tals

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13. WAVELET ATH

00.3/1000 population.

15. HTL A A TTT

la Malle sex
alt Merk
of peatood

| 7 Alcohol (10 or more grams per day)
or Diunetics

FObesity

#Hypertension

* Coranany heart disease

* Diabetes mellitus

* Chronic renal failure

fiHigh triglycerides

ooo © © ©

16. INTO NN]

e Tt affects both upper and lower limbs with:
attacks. Less often it presents with painful
tophaceous deposits (+ discharge) in Heben
and Bouchard's nodes|

a || # Most patients with hyperuricaemia ney
develop |gout and pouty patients may not he
hyperuricaemia at presentation,

la || # Patients can belover-excretens| of] uric a
nokmo-exereters or Underexcreters!

||| Fi Most cases of primary gout are due to
undersecretion.

alli Fewer than 10% are due to overprodue

17. by
eases

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—— ==
ee

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lity 0

A
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neutro

18. CUINA RATA

O Pain swelling. tendemess and increased) tel
the first metatarsophalangeal joint!

O Attacks ¡can be provoked by Surgery. traum:

© Mild attacks resolve with in 2) days.

0 More severe attacks take |7 10 10 days to re

19. adi with

o Old. frail taking thtazide diuretics e

21. O Tophi can oecun in long standing cases of
© 10% cases Kidney may be affected!

O HIN and e/e renal failure,

o Renal ¡stones can occur in 1094 cases)

O Renal disease is the major cause of death 1

22. INVESTIGA TONE

+ typical presentations such as inflammation of the!
metatarsophalangeal joint (also known as poda gr:
hyperurigaemia,

a Demonstration of monosodium urate (MSU)|
in synovial Avid or tophi continns the diagnosis lo

ot Since gout can present atypieally examine all
samples ofisynovial Mud aspirated from) jornts for
orystals, even lit not inflamed at the time,

1 | Gram staining and culture of a li fluid sI
be done, even ik MSU! crystals are found) since go
SEPSIS Can CO-EXISL

la ||| Fastine glucose and lipids should be perform
rule out hyperelytaemia and hyperlipidaemia asp
commonly associated with metabolic syndrome,

23. o * Renal une acid secretion (as detected by
hor urine sample) helpful in| diagnosis, in
patients with a family] history of young ont
gout, patients whose first attack of gout we
Unden|the age of 28. and patients with rena
stones. Such patients are likely to be avert
excreters of uric acid,

24. o Although a raised serum) uric acid level is
important risk factor for eout! the use of se
uric acid as a diagnostic) test is limited, It e
normal during acute gout, whilst patients y
hyperuricaemia may never develop ain atta
Studies sugeest that the cut-off point above
which a level can|be considered raised is 3
umoll.

27. lat
ape
hat
“POs

:

his
i

|
sing

|

vb
|

e il
ind as

morphit and

5

29. AS TRAIN TT

O INDOMBETHACIN(200me/day).

© Ideally pt should have only one attack of à

O lt can be achieved by xanthine oxidase
inhıbiton(allopurinol 300me/day),

fo Dose is according to serial plasma unie aci
level,

e First 3 months add a small dose of indome
to prevent ale attacks.

31. SSA.
kal

oe
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Ze
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= à
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tí

zn
ge:
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050 al
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en

32. NH RE EA PRIA KINN A

"¡Renal disease
"| [Renal colic.

«| [Chronic urate nephropathy results from
widespread deposition of urate orystals) in
interstitium of medulla and pyramids causi
inflammation) and fibrosis. End stage renal
failure ocewrs in up to 25% of cases of unt
chronic tophaceous gout,

33. © |Gout patients who have a 24-hour urinary
excretion of urie acid above 1100 me have
30% risk of developing urate and oxalate I
stones. Those with a measured urate exerel
greater than $00 me per 24 hours may ben
from allopurinol prophylaxis to prevent ur
nephropathy,

34. allcord imp i

.

nt spin

35. PLACA ALI Ce
THROAT YI CHONDRODA

37. EA UNA PA AT

o Dehydration

© lIntercurrent illness

e |Hyperparathyroidism.

O |long term use olf steroids
o |Hypathyroidism

o Any cause of arthritis

© Haemochromatosis

© | Wilson's! disease

© ¡Acromegaly
© |Dialysis
© Surgery or trauma

38. UNV ESTATE TINA

o Cirystals|have gotlaicharacteristic appearan
Under polarising microscope with weak
bireftinges.

o Radiograph shows caloification im both fib
hyaline cartilage,

40. flocal Ste
tó cont!

()
he

|

|
ation and instil
aci i) and pheny uUtazo

$ pre ent recurrence,

t aspi

i

attac

MANAGE
ia

| oJo
In

41. resolve) wi
progless

|
ula
oral intl
ill also bed

10

je

ts de

CS) US!

42. DURE A D ATL OL. HP EN CR

o [Acute cout

O |Septic arthritis

o |Osteoarthritis

o Rheumatoid arthritis

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44. is called milwaukee Ä
harge of the a
the periarticul

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due to

in
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45. THANK
WU

47. Psoriatic arthritis

HAMIDREZA SADEGHI

48. POTT’S SPINE
> ae

|
D. a
LA + ARE Pa
AN b ce =

Kritika Singh
38

49. CONGENITAL TALIPES
EQUINO VARUS

5

50. Ankylosing Spondylitis

Presented
By

MD. MONSUR RAHMAN

MM INSTITUTE OF PHYSIOTHERAPY & REHABILITATION
MULLANA, AMBALA

MPT (Musculoskeletal Disorders)

a

53. DR. KTARUN RAO
PG IN DEPT OF ORTHOPEDICS
CAIMS, KARIMNAGAR.